Cancers adapt to their mutational load by buffering protein misfolding stress

In asexual populations that don’t undergo recombination, such as cancer, deleterious mutations are expected to accrue readily due to genome-wide linkage between mutations. Despite this mutational load of often thousands of deleterious mutations, many tumors thrive. How tumors survive the damaging co...

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Bibliographic Details
Main Authors: Susanne Tilk, Judith Frydman, Christina Curtis, Dmitri A Petrov
Format: Article
Language:English
Published: eLife Sciences Publications Ltd 2024-11-01
Series:eLife
Subjects:
Online Access:https://elifesciences.org/articles/87301
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