EMCV VP2 degrades IFI16 through Caspase-dependent apoptosis to evade IFI16-STING pathway

Abstract Interferon (IFN)-γ inducible protein 16 (IFI16), a key DNA sensor, triggers downstream STING-dependent type I interferon (IFN-I) production and antiviral immunity. However, how the IFI16-STING signaling pathway is regulated by EMCV infection is still not well elucidated. In this study, we i...

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Main Authors: Ruofei Feng, Dianyu Li, Zhenfang Yan, Xiangrong Li, Jingying Xie
Format: Article
Language:English
Published: BMC 2024-11-01
Series:Virology Journal
Subjects:
Online Access:https://doi.org/10.1186/s12985-024-02568-8
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author Ruofei Feng
Dianyu Li
Zhenfang Yan
Xiangrong Li
Jingying Xie
author_facet Ruofei Feng
Dianyu Li
Zhenfang Yan
Xiangrong Li
Jingying Xie
author_sort Ruofei Feng
collection DOAJ
description Abstract Interferon (IFN)-γ inducible protein 16 (IFI16), a key DNA sensor, triggers downstream STING-dependent type I interferon (IFN-I) production and antiviral immunity. However, how the IFI16-STING signaling pathway is regulated by EMCV infection is still not well elucidated. In this study, we investigated the interaction between IFI16 and EMCV. Results indicated EMCV infection suppressed IFI16 expression in A549 cells. This study reveals that IFI16 plays an active role in combating EMCV. Screening viral proteins in conjunction with IFI16, we found that the EMCV VP2 protein hinders the antiviral response mediated by IFI16 by causing degradation of the IFI16 protein via the caspase-dependent apoptosis pathway. Our study communicates the antiviral role of the IFI16-STING pathway during EMCV infection. Importantly, this study unveils the novel mechanism by which VP2 counteracts the innate immune signaling activated by foreign DNA.
format Article
id doaj-art-ea256fe9d7064febb7e8adc4c1a9db1c
institution Kabale University
issn 1743-422X
language English
publishDate 2024-11-01
publisher BMC
record_format Article
series Virology Journal
spelling doaj-art-ea256fe9d7064febb7e8adc4c1a9db1c2024-11-24T12:10:37ZengBMCVirology Journal1743-422X2024-11-0121111710.1186/s12985-024-02568-8EMCV VP2 degrades IFI16 through Caspase-dependent apoptosis to evade IFI16-STING pathwayRuofei Feng0Dianyu Li1Zhenfang Yan2Xiangrong Li3Jingying Xie4Key Laboratory of Biotechnology and Bioengineering of State Ethnic Biomedical Research Center, Northwest Minzu UniversityKey Laboratory of Biotechnology and Bioengineering of State Ethnic Biomedical Research Center, Northwest Minzu UniversityKey Laboratory of Biotechnology and Bioengineering of State Ethnic Biomedical Research Center, Northwest Minzu UniversityKey Laboratory of Biotechnology and Bioengineering of State Ethnic Biomedical Research Center, Northwest Minzu UniversityKey Laboratory of Biotechnology and Bioengineering of State Ethnic Biomedical Research Center, Northwest Minzu UniversityAbstract Interferon (IFN)-γ inducible protein 16 (IFI16), a key DNA sensor, triggers downstream STING-dependent type I interferon (IFN-I) production and antiviral immunity. However, how the IFI16-STING signaling pathway is regulated by EMCV infection is still not well elucidated. In this study, we investigated the interaction between IFI16 and EMCV. Results indicated EMCV infection suppressed IFI16 expression in A549 cells. This study reveals that IFI16 plays an active role in combating EMCV. Screening viral proteins in conjunction with IFI16, we found that the EMCV VP2 protein hinders the antiviral response mediated by IFI16 by causing degradation of the IFI16 protein via the caspase-dependent apoptosis pathway. Our study communicates the antiviral role of the IFI16-STING pathway during EMCV infection. Importantly, this study unveils the novel mechanism by which VP2 counteracts the innate immune signaling activated by foreign DNA.https://doi.org/10.1186/s12985-024-02568-8EMCVIFI16Viral protein VP2Caspase-dependent apoptosis
spellingShingle Ruofei Feng
Dianyu Li
Zhenfang Yan
Xiangrong Li
Jingying Xie
EMCV VP2 degrades IFI16 through Caspase-dependent apoptosis to evade IFI16-STING pathway
Virology Journal
EMCV
IFI16
Viral protein VP2
Caspase-dependent apoptosis
title EMCV VP2 degrades IFI16 through Caspase-dependent apoptosis to evade IFI16-STING pathway
title_full EMCV VP2 degrades IFI16 through Caspase-dependent apoptosis to evade IFI16-STING pathway
title_fullStr EMCV VP2 degrades IFI16 through Caspase-dependent apoptosis to evade IFI16-STING pathway
title_full_unstemmed EMCV VP2 degrades IFI16 through Caspase-dependent apoptosis to evade IFI16-STING pathway
title_short EMCV VP2 degrades IFI16 through Caspase-dependent apoptosis to evade IFI16-STING pathway
title_sort emcv vp2 degrades ifi16 through caspase dependent apoptosis to evade ifi16 sting pathway
topic EMCV
IFI16
Viral protein VP2
Caspase-dependent apoptosis
url https://doi.org/10.1186/s12985-024-02568-8
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AT dianyuli emcvvp2degradesifi16throughcaspasedependentapoptosistoevadeifi16stingpathway
AT zhenfangyan emcvvp2degradesifi16throughcaspasedependentapoptosistoevadeifi16stingpathway
AT xiangrongli emcvvp2degradesifi16throughcaspasedependentapoptosistoevadeifi16stingpathway
AT jingyingxie emcvvp2degradesifi16throughcaspasedependentapoptosistoevadeifi16stingpathway