MGAT4A/Galectin9‐Driven N‐Glycosylation Aberration as a Promoting Mechanism for Poor Prognosis of Endometrial Cancer with TP53 Mutation

MGAT4A/Galectin9‐Driven N‐Glycosylation Aberration as a Promoting Mechanism for Poor Prognosis of Endometrial Cancer with TP53 Mutation

Abstract Emerging evidence recognizes aberrant glycosylation as the malignant characteristics of cancer cells, but little is known about glycogenes’ roles in endometrial carcinoma (EC), especially the most aggressive subtype carrying TP53 mutations. Using unsupervised hierarchical clustering, an 11‐...

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Main Authors: Zhen Zhu, Jingya Sun, Weiqing Xu, Qinghe Zeng, Hanyi Feng, Lijuan Zang, Yinyan He, Xiao He, Na Sheng, Xuelian Ren, Guobin Liu, He Huang, Ruimin Huang, Jun Yan
Format: Article
Language:English
Published: Wiley 2024-12-01
Series:Advanced Science
Subjects:
endometrial cancer
glucose metabolism
MGAT4A
N‐glycosylation
TP53 mutation
Online Access:https://doi.org/10.1002/advs.202409764
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https://doi.org/10.1002/advs.202409764

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