Axin2 coupled excessive Wnt‐glycolysis signaling mediates social defect in autism spectrum disorders

Axin2 coupled excessive Wnt‐glycolysis signaling mediates social defect in autism spectrum disorders

Abstract Social dysfunction is the core syndrome of autism spectrum disorder (ASD) and lacks effective medicine. Although numerous risk genes and relevant environmental factors have been identified, the convergent molecular mechanism underlying ASD‐associated social dysfunction remains largely elusi...

Full description

Saved in:
Bibliographic Details
Main Authors: Mengmeng Wang, Panpan Xian, Weian Zheng, Zhenzhen Li, Andi Chen, Haoxiang Xiao, Chao Xu, Fei Wang, Honghui Mao, Han Meng, Youyi Zhao, Ceng Luo, Yazhou Wang, Shengxi Wu
Format: Article
Language:English
Published: Springer Nature 2023-04-01
Series:EMBO Molecular Medicine
Subjects:
Axin2
glycolysis
social dysfunction
Wnt signaling
Online Access:https://doi.org/10.15252/emmm.202217101
Tags: Add Tag
No Tags, Be the first to tag this record!

Internet

https://doi.org/10.15252/emmm.202217101

Similar Items