An Exogenous NO Donor Provokes Mechanical Alternans in Normal Rat Atria and Impairs Sarcomere Contractility in Right Atrial Cardiomyocytes in Atrial Fibrillation

An Exogenous NO Donor Provokes Mechanical Alternans in Normal Rat Atria and Impairs Sarcomere Contractility in Right Atrial Cardiomyocytes in Atrial Fibrillation

Atrial fibrillation (AF) is the most common arrhythmia worldwide. AF is associated with a deficiency in nitric oxide (NO) production, which contributes to disturbances in the electrical and mechanical function of the atrial myocardium. NO donors are considered promising for the treatment and prevent...

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Bibliographic Details
Main Authors: Xenia Butova, Tatiana Myachina, Polina Mikhryakova, Raisa Simonova, Daniil Shchepkin, Anastasia Khokhlova
Format: Article
Language:English
Published: MDPI AG 2025-05-01
Series:Biomolecules
Subjects:
nitric oxide
NO donor
atrial fibrillation
left and right atria
cardiomyocyte contractility
mechanical alternans
Online Access:https://www.mdpi.com/2218-273X/15/5/735
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