Fibrinogen and Neuroinflammation in the Neurovascular Unit in Stroke

Yi Chen,1 Lu Han,1 De-Sheng Zhu,1,2 Yang-Tai Guan1,3 1Department of Neurology, Ren Ji Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, 200127, People’s Republic of China; 2Department of Neurology, Baoshan Branch, Renji Hospital, Shanghai Jiao Tong University School of Medicine,...

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Main Authors: Chen Y, Han L, Zhu DS, Guan YT
Format: Article
Language:English
Published: Dove Medical Press 2025-04-01
Series:Journal of Inflammation Research
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Online Access:https://www.dovepress.com/fibrinogen-and-neuroinflammation-in-the-neurovascular-unit-in-stroke-peer-reviewed-fulltext-article-JIR
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author Chen Y
Han L
Zhu DS
Guan YT
author_facet Chen Y
Han L
Zhu DS
Guan YT
author_sort Chen Y
collection DOAJ
description Yi Chen,1 Lu Han,1 De-Sheng Zhu,1,2 Yang-Tai Guan1,3 1Department of Neurology, Ren Ji Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, 200127, People’s Republic of China; 2Department of Neurology, Baoshan Branch, Renji Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, 200444, People’s Republic of China; 3Department of Neurology, Punan Hospital, Shanghai, 200125, People’s Republic of ChinaCorrespondence: Yang-Tai Guan; De-Sheng Zhu, Department of Neurology, Ren Ji Hospital, Shanghai Jiao Tong University School of Medicine, No. 160, Pujian Road, Shanghai, 200127, People’s Republic of China, Tel +86-13386271865 ; +86-13564719779, Fax +86-21-68383482, Email yangtaiguan@sina.com; deshengzhu2008@sina.comAbstract: Stroke remains a leading cause of death and disability worldwide. Recent evidence suggests that stroke pathophysiology extends beyond vascular dysfunction to include complex interactions within the neurovascular unit (NVU), particularly involving fibrinogen. This blood-derived protein accumulates in the brain following blood-brain barrier (BBB) disruption and plays crucial roles in neuroinflammation and tissue repair. Through its unique structural domains, fibrinogen interacts with multiple cellular components, including astrocytes, microglia, and neural stem cells, thereby modulating inflammatory responses and neural repair mechanisms. This review examines fibrinogen’s structure and its diverse functions in stroke pathophysiology, focusing on its interactions with vascular cells, glial cells, and peripheral immune cells. We also discuss emerging therapeutic strategies targeting fibrinogen-mediated pathways and the challenge of translating experimental results into effective clinical treatments. Keywords: fibrinogen, neuroinflammation, stroke, neurovascular unit, neurological disorders
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spelling doaj-art-98bbebd0e31e4209bd7da0ff1f20b75c2025-08-20T03:44:35ZengDove Medical PressJournal of Inflammation Research1178-70312025-04-01Volume 1845674584101664Fibrinogen and Neuroinflammation in the Neurovascular Unit in StrokeChen YHan LZhu DSGuan YTYi Chen,1 Lu Han,1 De-Sheng Zhu,1,2 Yang-Tai Guan1,3 1Department of Neurology, Ren Ji Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, 200127, People’s Republic of China; 2Department of Neurology, Baoshan Branch, Renji Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, 200444, People’s Republic of China; 3Department of Neurology, Punan Hospital, Shanghai, 200125, People’s Republic of ChinaCorrespondence: Yang-Tai Guan; De-Sheng Zhu, Department of Neurology, Ren Ji Hospital, Shanghai Jiao Tong University School of Medicine, No. 160, Pujian Road, Shanghai, 200127, People’s Republic of China, Tel +86-13386271865 ; +86-13564719779, Fax +86-21-68383482, Email yangtaiguan@sina.com; deshengzhu2008@sina.comAbstract: Stroke remains a leading cause of death and disability worldwide. Recent evidence suggests that stroke pathophysiology extends beyond vascular dysfunction to include complex interactions within the neurovascular unit (NVU), particularly involving fibrinogen. This blood-derived protein accumulates in the brain following blood-brain barrier (BBB) disruption and plays crucial roles in neuroinflammation and tissue repair. Through its unique structural domains, fibrinogen interacts with multiple cellular components, including astrocytes, microglia, and neural stem cells, thereby modulating inflammatory responses and neural repair mechanisms. This review examines fibrinogen’s structure and its diverse functions in stroke pathophysiology, focusing on its interactions with vascular cells, glial cells, and peripheral immune cells. We also discuss emerging therapeutic strategies targeting fibrinogen-mediated pathways and the challenge of translating experimental results into effective clinical treatments. Keywords: fibrinogen, neuroinflammation, stroke, neurovascular unit, neurological disordershttps://www.dovepress.com/fibrinogen-and-neuroinflammation-in-the-neurovascular-unit-in-stroke-peer-reviewed-fulltext-article-JIRfibrinogenneuroinflammationstrokeneurovascular unitneurological disorders
spellingShingle Chen Y
Han L
Zhu DS
Guan YT
Fibrinogen and Neuroinflammation in the Neurovascular Unit in Stroke
Journal of Inflammation Research
fibrinogen
neuroinflammation
stroke
neurovascular unit
neurological disorders
title Fibrinogen and Neuroinflammation in the Neurovascular Unit in Stroke
title_full Fibrinogen and Neuroinflammation in the Neurovascular Unit in Stroke
title_fullStr Fibrinogen and Neuroinflammation in the Neurovascular Unit in Stroke
title_full_unstemmed Fibrinogen and Neuroinflammation in the Neurovascular Unit in Stroke
title_short Fibrinogen and Neuroinflammation in the Neurovascular Unit in Stroke
title_sort fibrinogen and neuroinflammation in the neurovascular unit in stroke
topic fibrinogen
neuroinflammation
stroke
neurovascular unit
neurological disorders
url https://www.dovepress.com/fibrinogen-and-neuroinflammation-in-the-neurovascular-unit-in-stroke-peer-reviewed-fulltext-article-JIR
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AT hanl fibrinogenandneuroinflammationintheneurovascularunitinstroke
AT zhuds fibrinogenandneuroinflammationintheneurovascularunitinstroke
AT guanyt fibrinogenandneuroinflammationintheneurovascularunitinstroke