Showing 21 - 40 results of 116 for search '"idiopathic pulmonary fibrosis"', query time: 0.09s Refine Results
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    A scoping review of the unmet needs of patients diagnosed with idiopathic pulmonary fibrosis (IPF). by Carita Bramhill, Donna Langan, Helen Mulryan, Jessica Eustace-Cook, Anne-Marie Russell, Anne-Marie Brady

    Published 2024-01-01
    “…<h4>Aims</h4>Patients diagnosed with idiopathic pulmonary fibrosis (IPF) have a high symptom burden and numerous needs that remain largely unaddressed despite advances in available treatment options. …”
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    Does Technetium-99m Diethylenetriaminepentaacetate Clearance Predict the Clinical Course of Idiopathic Pulmonary Fibrosis? by Marco Mura, Gelorma Belmonte, Stefano Fanti, Angela Maria Pacilli, Luca Fasano, Maurizio Zompatori, Mario Schiavina, Mario Fabbri

    Published 2004-01-01
    “…Clearance of inhaled technetium-99m diethylenetriaminepentaacetate (99mTc-DTPA) is a potential indicator of disease activity and progression in idiopathic pulmonary fibrosis (IPF). The objective of the present study was to evaluate the prognostic value of 99mTc-DTPA scans in IPF. …”
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    Different Levels of Autophagy Activity in Mesenchymal Stem Cells Are Involved in the Progression of Idiopathic Pulmonary Fibrosis by Hongxia Tao, Qin Lv, Jing Zhang, Lijuan Chen, Yang Yang, Wei Sun

    Published 2024-01-01
    “…Idiopathic pulmonary fibrosis (IPF) is an age-related lung interstitial disease that occurs predominantly in people over 65 years of age and for which there is a lack of effective therapeutic agents. …”
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    Distinct Roles of Wnt/β-Catenin Signaling in the Pathogenesis of Chronic Obstructive Pulmonary Disease and Idiopathic Pulmonary Fibrosis by Juan Shi, Feng Li, Meihui Luo, Jun Wei, Xiaoming Liu

    Published 2017-01-01
    “…Increasing lines of evidence recently demonstrated that a dysregulated activation of Wnt signaling, particularly the Wnt/β-catenin signaling, was involved in the pathogenesis of chronic pulmonary diseases, such as chronic obstructive pulmonary disease (COPD) and idiopathic pulmonary fibrosis (IPF). In this respect, Wnt signaling interacts with other cellular signaling pathways to regulate the initiation and pathogenic procedures of airway inflammation and remodeling, pulmonary myofibroblast proliferation, epithelial-to-mesenchymal transition (EMT), and development of emphysema. …”
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