Salidroside impedes Ang II-infused myocardial fibrosis by activating the SIRT1-Nrf2 pathway

Objective(s): This research examined the protective function of salidroside (SAL) against angiotensin II (Ang II)-infused myocardial fibrosis and its associated mechanism.Materials and Methods: The C57BL/6 male murine models (n=24) received either saline solution or Ang II (1500 ng/kg/day) subcutane...

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Main Authors: Xi Zhu, Zhen Hai, Zhongping Ning
Format: Article
Language:English
Published: Mashhad University of Medical Sciences 2025-06-01
Series:Iranian Journal of Basic Medical Sciences
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Online Access:https://ijbms.mums.ac.ir/article_25729_ac5a2b9b6bc377109be13f1e22f4e0e3.pdf
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author Xi Zhu
Zhen Hai
Zhongping Ning
author_facet Xi Zhu
Zhen Hai
Zhongping Ning
author_sort Xi Zhu
collection DOAJ
description Objective(s): This research examined the protective function of salidroside (SAL) against angiotensin II (Ang II)-infused myocardial fibrosis and its associated mechanism.Materials and Methods: The C57BL/6 male murine models (n=24) received either saline solution or Ang II (1500 ng/kg/day) subcutaneously and an oral dosage of SAL (50 mg/kg/day) once daily for 28 days. Newborn Sprague-Dawley (SD) rats were used to isolate atrial fibroblasts.Results: The fibrotic region was raised by Ang II infusion, while SAL treatment inhibited it. Collagen I and III expression was raised by Ang II induction, but SAL therapy reduced their expression. SAL therapy also decreased the expression of other fibroblast differentiation-related markers induced by Ang II infusion. It elevated SIRT1, Nrf2, and HO-1 levels in atrial fibroblasts. Additionally, SAL significantly inhibited atrial fibroblasts, whereas EX527, an inhibitor of SIRT1, noticeably increased the migration ability. Furthermore, SAL suppressed intracellular ROS production and oxidative stress in Ang II-infused atrial fibroblasts. Conclusion: SAL protects against myocardial fibrosis infused by Ang II by activating the SIRT1-Nrf2 pathway.
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institution Kabale University
issn 2008-3866
2008-3874
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publishDate 2025-06-01
publisher Mashhad University of Medical Sciences
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series Iranian Journal of Basic Medical Sciences
spelling doaj-art-db3b478dbc304d8381b9a934dfe73aaa2025-08-20T03:56:45ZengMashhad University of Medical SciencesIranian Journal of Basic Medical Sciences2008-38662008-38742025-06-0128681582410.22038/ijbms.2025.83659.1810525729Salidroside impedes Ang II-infused myocardial fibrosis by activating the SIRT1-Nrf2 pathwayXi Zhu0Zhen Hai1Zhongping Ning2Department of Cardiology, Shanghai Pudong New Area Zhoupu Hospital (Shanghai Health Medical College Affiliated Zhoupu Hospital) Shanghai 201318, ChinaDepartment of Cardiology, Shanghai Pudong New Area Zhoupu Hospital (Shanghai Health Medical College Affiliated Zhoupu Hospital) Shanghai 201318, ChinaDepartment of Cardiology, Shanghai Pudong New Area Zhoupu Hospital (Shanghai Health Medical College Affiliated Zhoupu Hospital) Shanghai 201318, ChinaObjective(s): This research examined the protective function of salidroside (SAL) against angiotensin II (Ang II)-infused myocardial fibrosis and its associated mechanism.Materials and Methods: The C57BL/6 male murine models (n=24) received either saline solution or Ang II (1500 ng/kg/day) subcutaneously and an oral dosage of SAL (50 mg/kg/day) once daily for 28 days. Newborn Sprague-Dawley (SD) rats were used to isolate atrial fibroblasts.Results: The fibrotic region was raised by Ang II infusion, while SAL treatment inhibited it. Collagen I and III expression was raised by Ang II induction, but SAL therapy reduced their expression. SAL therapy also decreased the expression of other fibroblast differentiation-related markers induced by Ang II infusion. It elevated SIRT1, Nrf2, and HO-1 levels in atrial fibroblasts. Additionally, SAL significantly inhibited atrial fibroblasts, whereas EX527, an inhibitor of SIRT1, noticeably increased the migration ability. Furthermore, SAL suppressed intracellular ROS production and oxidative stress in Ang II-infused atrial fibroblasts. Conclusion: SAL protects against myocardial fibrosis infused by Ang II by activating the SIRT1-Nrf2 pathway.https://ijbms.mums.ac.ir/article_25729_ac5a2b9b6bc377109be13f1e22f4e0e3.pdfangiotensin iioxidative stressreactive oxygen speciessalidrosidesirt1
spellingShingle Xi Zhu
Zhen Hai
Zhongping Ning
Salidroside impedes Ang II-infused myocardial fibrosis by activating the SIRT1-Nrf2 pathway
Iranian Journal of Basic Medical Sciences
angiotensin ii
oxidative stress
reactive oxygen species
salidroside
sirt1
title Salidroside impedes Ang II-infused myocardial fibrosis by activating the SIRT1-Nrf2 pathway
title_full Salidroside impedes Ang II-infused myocardial fibrosis by activating the SIRT1-Nrf2 pathway
title_fullStr Salidroside impedes Ang II-infused myocardial fibrosis by activating the SIRT1-Nrf2 pathway
title_full_unstemmed Salidroside impedes Ang II-infused myocardial fibrosis by activating the SIRT1-Nrf2 pathway
title_short Salidroside impedes Ang II-infused myocardial fibrosis by activating the SIRT1-Nrf2 pathway
title_sort salidroside impedes ang ii infused myocardial fibrosis by activating the sirt1 nrf2 pathway
topic angiotensin ii
oxidative stress
reactive oxygen species
salidroside
sirt1
url https://ijbms.mums.ac.ir/article_25729_ac5a2b9b6bc377109be13f1e22f4e0e3.pdf
work_keys_str_mv AT xizhu salidrosideimpedesangiiinfusedmyocardialfibrosisbyactivatingthesirt1nrf2pathway
AT zhenhai salidrosideimpedesangiiinfusedmyocardialfibrosisbyactivatingthesirt1nrf2pathway
AT zhongpingning salidrosideimpedesangiiinfusedmyocardialfibrosisbyactivatingthesirt1nrf2pathway