Cyclin dependent kinase 9 inhibitor induces transcription-replication conflicts and DNA damage accumulation in breast cancer

Abstract Background Cyclin-dependent kinase 9 (CDK9) is a crucial regulator of transcriptional progression of RNA polymerase-II (RNAP2). RNA polymerases trapped in DNA can be a source of transcription-replication conflict (T-R conflict), which is a common source of replication stress. AZD4573, a hig...

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Main Authors: Minyoung Lee, Kyung-Hun Lee, Ahrum Min, So Hyeon Kim, Sujin Ham, Hae Min Hwang, Youlim Noh, Yu-Jin Kim, Dae-Won Lee, Jiwon Koh, Seock-Ah Im
Format: Article
Language:English
Published: BMC 2025-07-01
Series:Cancer Cell International
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Online Access:https://doi.org/10.1186/s12935-025-03897-6
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author Minyoung Lee
Kyung-Hun Lee
Ahrum Min
So Hyeon Kim
Sujin Ham
Hae Min Hwang
Youlim Noh
Yu-Jin Kim
Dae-Won Lee
Jiwon Koh
Seock-Ah Im
author_facet Minyoung Lee
Kyung-Hun Lee
Ahrum Min
So Hyeon Kim
Sujin Ham
Hae Min Hwang
Youlim Noh
Yu-Jin Kim
Dae-Won Lee
Jiwon Koh
Seock-Ah Im
author_sort Minyoung Lee
collection DOAJ
description Abstract Background Cyclin-dependent kinase 9 (CDK9) is a crucial regulator of transcriptional progression of RNA polymerase-II (RNAP2). RNA polymerases trapped in DNA can be a source of transcription-replication conflict (T-R conflict), which is a common source of replication stress. AZD4573, a highly selective CDK9 inhibitor, has been shown to induce apoptosis in leukemia cell lines, while its anti-tumor potential in breast cancer has yet to be elucidated. Methods To evaluate the cytotoxicity of AZD4573 in vitro, MTT assays were performed. The expression of signal transduction molecules was determined using Western blotting, immunoprecipitation, and immunofluorescence. Apoptotic cell death was verified by the annexin-V assay. DNA strand breaks and repair efficacy were evaluated through the alkaline comet assay. The siRNA knock-down system was used to confirm the action mechanism. Results AZD4573 induced T-R conflicts during S-phase, increasing replication stress and DNA strand breaks, resulting in apoptosis by induction of caspase-3. Furthermore, we identified Dead-box 25 (DDX25) helicase as a key mediator in resolving the T-R conflicts. Nuclear translocation of DDX25 correlated with reduced sensitivity to AZD4573 by the resolution of T-R conflicts. Conclusions Inhibition of CDK9 by AZD4573 induces the accumulation of DNA damage through T-R conflicts. DDX25 helicases were identified as a key mediator in resolving T-R conflicts and the reduced sensitivity to AZD4573.
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series Cancer Cell International
spelling doaj-art-d98e1e4b87a44e3a86af5b7de8e176152025-08-20T03:46:16ZengBMCCancer Cell International1475-28672025-07-0125111410.1186/s12935-025-03897-6Cyclin dependent kinase 9 inhibitor induces transcription-replication conflicts and DNA damage accumulation in breast cancerMinyoung Lee0Kyung-Hun Lee1Ahrum Min2So Hyeon Kim3Sujin Ham4Hae Min Hwang5Youlim Noh6Yu-Jin Kim7Dae-Won Lee8Jiwon Koh9Seock-Ah Im10Cancer Research Institute, Seoul National UniversityCancer Research Institute, Seoul National UniversityCancer Research Institute, Seoul National UniversityCancer Research Institute, Seoul National UniversityCancer Research Institute, Seoul National UniversityCancer Research Institute, Seoul National UniversityCancer Research Institute, Seoul National UniversityCancer Research Institute, Seoul National UniversityCancer Research Institute, Seoul National UniversityCancer Research Institute, Seoul National UniversityCancer Research Institute, Seoul National UniversityAbstract Background Cyclin-dependent kinase 9 (CDK9) is a crucial regulator of transcriptional progression of RNA polymerase-II (RNAP2). RNA polymerases trapped in DNA can be a source of transcription-replication conflict (T-R conflict), which is a common source of replication stress. AZD4573, a highly selective CDK9 inhibitor, has been shown to induce apoptosis in leukemia cell lines, while its anti-tumor potential in breast cancer has yet to be elucidated. Methods To evaluate the cytotoxicity of AZD4573 in vitro, MTT assays were performed. The expression of signal transduction molecules was determined using Western blotting, immunoprecipitation, and immunofluorescence. Apoptotic cell death was verified by the annexin-V assay. DNA strand breaks and repair efficacy were evaluated through the alkaline comet assay. The siRNA knock-down system was used to confirm the action mechanism. Results AZD4573 induced T-R conflicts during S-phase, increasing replication stress and DNA strand breaks, resulting in apoptosis by induction of caspase-3. Furthermore, we identified Dead-box 25 (DDX25) helicase as a key mediator in resolving the T-R conflicts. Nuclear translocation of DDX25 correlated with reduced sensitivity to AZD4573 by the resolution of T-R conflicts. Conclusions Inhibition of CDK9 by AZD4573 induces the accumulation of DNA damage through T-R conflicts. DDX25 helicases were identified as a key mediator in resolving T-R conflicts and the reduced sensitivity to AZD4573.https://doi.org/10.1186/s12935-025-03897-6Breast cancerCyclin dependent kinase 9DNA damageTranscription-replication conflict
spellingShingle Minyoung Lee
Kyung-Hun Lee
Ahrum Min
So Hyeon Kim
Sujin Ham
Hae Min Hwang
Youlim Noh
Yu-Jin Kim
Dae-Won Lee
Jiwon Koh
Seock-Ah Im
Cyclin dependent kinase 9 inhibitor induces transcription-replication conflicts and DNA damage accumulation in breast cancer
Cancer Cell International
Breast cancer
Cyclin dependent kinase 9
DNA damage
Transcription-replication conflict
title Cyclin dependent kinase 9 inhibitor induces transcription-replication conflicts and DNA damage accumulation in breast cancer
title_full Cyclin dependent kinase 9 inhibitor induces transcription-replication conflicts and DNA damage accumulation in breast cancer
title_fullStr Cyclin dependent kinase 9 inhibitor induces transcription-replication conflicts and DNA damage accumulation in breast cancer
title_full_unstemmed Cyclin dependent kinase 9 inhibitor induces transcription-replication conflicts and DNA damage accumulation in breast cancer
title_short Cyclin dependent kinase 9 inhibitor induces transcription-replication conflicts and DNA damage accumulation in breast cancer
title_sort cyclin dependent kinase 9 inhibitor induces transcription replication conflicts and dna damage accumulation in breast cancer
topic Breast cancer
Cyclin dependent kinase 9
DNA damage
Transcription-replication conflict
url https://doi.org/10.1186/s12935-025-03897-6
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