Genetic variants in the Fat and Obesity Associated (FTO) gene and risk of Alzheimer's disease.

<h4>Background</h4>Recent studies showed that polymorphisms in the Fat and Obesity-Associated (FTO) gene have robust effects on obesity, obesity-related traits and endophenotypes associated with Alzheimer's disease (AD).<h4>Methods</h4>We used 1,877 Caucasian cases and c...

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Main Authors: Christiane Reitz, Giuseppe Tosto, Richard Mayeux, Jose A Luchsinger, NIA-LOAD/NCRAD Family Study Group, Alzheimer's Disease Neuroimaging Initiative
Format: Article
Language:English
Published: Public Library of Science (PLoS) 2012-01-01
Series:PLoS ONE
Online Access:https://journals.plos.org/plosone/article/file?id=10.1371/journal.pone.0050354&type=printable
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author Christiane Reitz
Giuseppe Tosto
Richard Mayeux
Jose A Luchsinger
NIA-LOAD/NCRAD Family Study Group
Alzheimer's Disease Neuroimaging Initiative
author_facet Christiane Reitz
Giuseppe Tosto
Richard Mayeux
Jose A Luchsinger
NIA-LOAD/NCRAD Family Study Group
Alzheimer's Disease Neuroimaging Initiative
author_sort Christiane Reitz
collection DOAJ
description <h4>Background</h4>Recent studies showed that polymorphisms in the Fat and Obesity-Associated (FTO) gene have robust effects on obesity, obesity-related traits and endophenotypes associated with Alzheimer's disease (AD).<h4>Methods</h4>We used 1,877 Caucasian cases and controls from the NIA-LOAD study and 1,093 Caribbean Hispanics to further explore the association of FTO with AD. Using logistic regression, we assessed 42 SNPs in introns 1 and 2, the region previously reported to be associated with AD endophenotypes, which had been derived by genome-wide screenings. In addition, we performed gene expression analyses of neuropathologically confirmed AD cases and controls of two independent datasets (19 AD cases, 10 controls; 176 AD cases, 188 controls) using within- and between-group factors ANOVA of log(10) transformed rank invariant normalized expression data.<h4>Results</h4>In the NIALOAD study, one SNP was significantly associated with AD and three additional markers were close to significance (rs6499640, rs10852521, rs16945088, rs8044769, FDR p-value: 0.05<p<0.09). Two of the SNPs are in strong LD (D'>0.9) with the previously reported SNPs. In the Caribbean Hispanic dataset, we identified three SNPs (rs17219084, rs11075996, rs11075997, FDR p-value: 0.009<p<0.01) that were associated with AD. These results were confirmed by haplotype analyses and in a metaanalysis in which we included the ADNI dataset. FTO had a significantly lower expresssion in AD cases compared to controls in two independent datasets derived from human cortex and amygdala tissue, respectively (p = 2.18 × 10-5 and p<0.0001).<h4>Conclusions</h4>Our data support the notion that genetic variation in Introns 1 and 2 of the FTO gene may contribute to AD risk.
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spelling doaj-art-d7ffe3b8d3c04699bb2a64face9af7d72025-08-20T03:46:53ZengPublic Library of Science (PLoS)PLoS ONE1932-62032012-01-01712e5035410.1371/journal.pone.0050354Genetic variants in the Fat and Obesity Associated (FTO) gene and risk of Alzheimer's disease.Christiane ReitzGiuseppe TostoRichard MayeuxJose A LuchsingerNIA-LOAD/NCRAD Family Study GroupAlzheimer's Disease Neuroimaging Initiative<h4>Background</h4>Recent studies showed that polymorphisms in the Fat and Obesity-Associated (FTO) gene have robust effects on obesity, obesity-related traits and endophenotypes associated with Alzheimer's disease (AD).<h4>Methods</h4>We used 1,877 Caucasian cases and controls from the NIA-LOAD study and 1,093 Caribbean Hispanics to further explore the association of FTO with AD. Using logistic regression, we assessed 42 SNPs in introns 1 and 2, the region previously reported to be associated with AD endophenotypes, which had been derived by genome-wide screenings. In addition, we performed gene expression analyses of neuropathologically confirmed AD cases and controls of two independent datasets (19 AD cases, 10 controls; 176 AD cases, 188 controls) using within- and between-group factors ANOVA of log(10) transformed rank invariant normalized expression data.<h4>Results</h4>In the NIALOAD study, one SNP was significantly associated with AD and three additional markers were close to significance (rs6499640, rs10852521, rs16945088, rs8044769, FDR p-value: 0.05<p<0.09). Two of the SNPs are in strong LD (D'>0.9) with the previously reported SNPs. In the Caribbean Hispanic dataset, we identified three SNPs (rs17219084, rs11075996, rs11075997, FDR p-value: 0.009<p<0.01) that were associated with AD. These results were confirmed by haplotype analyses and in a metaanalysis in which we included the ADNI dataset. FTO had a significantly lower expresssion in AD cases compared to controls in two independent datasets derived from human cortex and amygdala tissue, respectively (p = 2.18 × 10-5 and p<0.0001).<h4>Conclusions</h4>Our data support the notion that genetic variation in Introns 1 and 2 of the FTO gene may contribute to AD risk.https://journals.plos.org/plosone/article/file?id=10.1371/journal.pone.0050354&type=printable
spellingShingle Christiane Reitz
Giuseppe Tosto
Richard Mayeux
Jose A Luchsinger
NIA-LOAD/NCRAD Family Study Group
Alzheimer's Disease Neuroimaging Initiative
Genetic variants in the Fat and Obesity Associated (FTO) gene and risk of Alzheimer's disease.
PLoS ONE
title Genetic variants in the Fat and Obesity Associated (FTO) gene and risk of Alzheimer's disease.
title_full Genetic variants in the Fat and Obesity Associated (FTO) gene and risk of Alzheimer's disease.
title_fullStr Genetic variants in the Fat and Obesity Associated (FTO) gene and risk of Alzheimer's disease.
title_full_unstemmed Genetic variants in the Fat and Obesity Associated (FTO) gene and risk of Alzheimer's disease.
title_short Genetic variants in the Fat and Obesity Associated (FTO) gene and risk of Alzheimer's disease.
title_sort genetic variants in the fat and obesity associated fto gene and risk of alzheimer s disease
url https://journals.plos.org/plosone/article/file?id=10.1371/journal.pone.0050354&type=printable
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