FTO Alleviates Hepatic Ischemia-Reperfusion Injury by Regulating Apoptosis and Autophagy
Conclusion: FTO could act as a protective effector during hepatic IRI, associated with decreased apoptosis and autophagy. FTO-mediated m6A demethylation modification may be an important therapeutic target for hepatic IRI.
Saved in:
Main Authors: | Pi-Xiao Wang, Ling Zhu, Mei Xiang, Rixin Zhang, Xiaolin Zheng, Zhi Zheng, Kai Li |
---|---|
Format: | Article |
Language: | English |
Published: |
Wiley
2025-01-01
|
Series: | Gastroenterology Research and Practice |
Online Access: | http://dx.doi.org/10.1155/grp/5587859 |
Tags: |
Add Tag
No Tags, Be the first to tag this record!
|
Similar Items
-
Alleviation of Hepatic Ischemia Reperfusion Injury by Oleanolic Acid Pretreating via Reducing HMGB1 Release and Inhibiting Apoptosis and Autophagy
by: Wenwen Wang, et al.
Published: (2019-01-01) -
Ethyl Pyruvate Ameliorates Hepatic Ischemia-Reperfusion Injury by Inhibiting Intrinsic Pathway of Apoptosis and Autophagy
by: Miao Shen, et al.
Published: (2013-01-01) -
Luteolin Pretreatment Attenuates Hepatic Ischemia-Reperfusion Injury in Mice by Inhibiting Inflammation, Autophagy, and Apoptosis via the ERK/PPARα Pathway
by: Yuhui Jiang, et al.
Published: (2022-01-01) -
Effect of Exercise Preconditioning on Autophagy and Apoptosis of Cardiomyocytes in Aged Rats with Myocardial Ischemia Reperfusion Injury
by: LI Hongyu, et al.
Published: (2022-04-01) -
Mitochondrial apoptosis in response to cardiac ischemia-reperfusion injury
by: Kaixin Wang, et al.
Published: (2025-01-01)