UV-C treatment for hepatitis A Virus inactivation in Ulva lactuca edible seaweed

The presence of hepatitis A virus (HAV) contamination stands as a notable contributing factor to acute hepatitis, sometimes leading to acute liver failure. HAV transmission is predominantly associated with the ingestion of water and food contaminated with the virus, as well as through sexual contact...

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Main Authors: Pantu Kumar Roy, Anamika Roy, Eun Bi Jeon, Shin Young Park
Format: Article
Language:English
Published: Elsevier 2024-12-01
Series:Applied Food Research
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Online Access:http://www.sciencedirect.com/science/article/pii/S2772502224002579
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author Pantu Kumar Roy
Anamika Roy
Eun Bi Jeon
Shin Young Park
author_facet Pantu Kumar Roy
Anamika Roy
Eun Bi Jeon
Shin Young Park
author_sort Pantu Kumar Roy
collection DOAJ
description The presence of hepatitis A virus (HAV) contamination stands as a notable contributing factor to acute hepatitis, sometimes leading to acute liver failure. HAV transmission is predominantly associated with the ingestion of water and food contaminated with the virus, as well as through sexual contact among men who engage in sexual activity. The effectiveness of UV-C on the viable reduction of a HAV strain on edible seaweed Ulva lactuca was tested at different conditions (control, 72, 216, 360, and 504 mWs/cm2 of UV-C dose) and the result of treatment on seaweed quality. To identify infectious and noninfectious virus components, the HAV was exposed using RT-qPCR with propidium monoazide/sarkosyl. HAV was decreased in seaweed when treatment with UV-C (control, 72, 216, 360, and 504 mWs/cm2) in non-PMA (0.94, 1.09, 1.67, and 2.32 log) and PMA + Sarkosyl-treated (1.03, 1.47, 1.94, and 3.02 log) reduction titers, respectively. The pH and Hunter color of the control and UV-C exposure seaweed were exhibited significant different (P > 0.05), while but sensory characteristics not significantly different. These results suggest the potential utility of PMA/RT-qPCR in identifying HAV infectivity without compromising the quality of seaweed following UV-C treatment. HAV can be inactivated in the food matrix at commercially satisfactory treatment of UV-C 504 mWs/cm2.
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spelling doaj-art-cfc79e5d2f304971aa7ed62566b1e2c52024-12-18T08:55:07ZengElsevierApplied Food Research2772-50222024-12-0142100647UV-C treatment for hepatitis A Virus inactivation in Ulva lactuca edible seaweedPantu Kumar Roy0Anamika Roy1Eun Bi Jeon2Shin Young Park3Institute of Marine Industry, Department of Seafood Science and Technology, Gyeongsang National University, Tongyeong 53064, Republic of KoreaInstitute of Marine Industry, Department of Seafood Science and Technology, Gyeongsang National University, Tongyeong 53064, Republic of KoreaInstitute of Marine Industry, Department of Seafood Science and Technology, Gyeongsang National University, Tongyeong 53064, Republic of KoreaCorresponding author: Department of Seafood Science and Technology, Gyeongsang National University, Tongyeong 53064, Republic of Korea.; Institute of Marine Industry, Department of Seafood Science and Technology, Gyeongsang National University, Tongyeong 53064, Republic of KoreaThe presence of hepatitis A virus (HAV) contamination stands as a notable contributing factor to acute hepatitis, sometimes leading to acute liver failure. HAV transmission is predominantly associated with the ingestion of water and food contaminated with the virus, as well as through sexual contact among men who engage in sexual activity. The effectiveness of UV-C on the viable reduction of a HAV strain on edible seaweed Ulva lactuca was tested at different conditions (control, 72, 216, 360, and 504 mWs/cm2 of UV-C dose) and the result of treatment on seaweed quality. To identify infectious and noninfectious virus components, the HAV was exposed using RT-qPCR with propidium monoazide/sarkosyl. HAV was decreased in seaweed when treatment with UV-C (control, 72, 216, 360, and 504 mWs/cm2) in non-PMA (0.94, 1.09, 1.67, and 2.32 log) and PMA + Sarkosyl-treated (1.03, 1.47, 1.94, and 3.02 log) reduction titers, respectively. The pH and Hunter color of the control and UV-C exposure seaweed were exhibited significant different (P > 0.05), while but sensory characteristics not significantly different. These results suggest the potential utility of PMA/RT-qPCR in identifying HAV infectivity without compromising the quality of seaweed following UV-C treatment. HAV can be inactivated in the food matrix at commercially satisfactory treatment of UV-C 504 mWs/cm2.http://www.sciencedirect.com/science/article/pii/S2772502224002579Hepatitis a virusPropidium monoazideSodium lauroyl sarcosinateSeaweeds Ulva LactucaUV-C
spellingShingle Pantu Kumar Roy
Anamika Roy
Eun Bi Jeon
Shin Young Park
UV-C treatment for hepatitis A Virus inactivation in Ulva lactuca edible seaweed
Applied Food Research
Hepatitis a virus
Propidium monoazide
Sodium lauroyl sarcosinate
Seaweeds Ulva Lactuca
UV-C
title UV-C treatment for hepatitis A Virus inactivation in Ulva lactuca edible seaweed
title_full UV-C treatment for hepatitis A Virus inactivation in Ulva lactuca edible seaweed
title_fullStr UV-C treatment for hepatitis A Virus inactivation in Ulva lactuca edible seaweed
title_full_unstemmed UV-C treatment for hepatitis A Virus inactivation in Ulva lactuca edible seaweed
title_short UV-C treatment for hepatitis A Virus inactivation in Ulva lactuca edible seaweed
title_sort uv c treatment for hepatitis a virus inactivation in ulva lactuca edible seaweed
topic Hepatitis a virus
Propidium monoazide
Sodium lauroyl sarcosinate
Seaweeds Ulva Lactuca
UV-C
url http://www.sciencedirect.com/science/article/pii/S2772502224002579
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