Heart over mind: metabolic control of white adipose tissue and liver

Abstract Increasing evidence suggests that the heart controls the metabolism of peripheral organs. Olson and colleagues previously demonstrated that miR‐208a controls systemic energy homeostasis through the regulation of MED13 in cardiomyocytes (Grueter et al, 2012). In their follow‐up study in this...

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Main Authors: Michinari Nakamura, Junichi Sadoshima
Format: Article
Language:English
Published: Springer Nature 2014-12-01
Series:EMBO Molecular Medicine
Online Access:https://doi.org/10.15252/emmm.201404749
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author Michinari Nakamura
Junichi Sadoshima
author_facet Michinari Nakamura
Junichi Sadoshima
author_sort Michinari Nakamura
collection DOAJ
description Abstract Increasing evidence suggests that the heart controls the metabolism of peripheral organs. Olson and colleagues previously demonstrated that miR‐208a controls systemic energy homeostasis through the regulation of MED13 in cardiomyocytes (Grueter et al, 2012). In their follow‐up study in this issue of EMBO Molecular Medicine, white adipose tissue (WAT) and liver are identified as the physiological targets of cardiac MED13 signaling, most likely through cardiac‐derived circulating factors, which boost energy consumption by upregulating metabolic gene expression and increasing mitochondrial numbers (Baskin et al, 2014). In turn, increased energy expenditure in WAT and the liver confers leanness. These findings strengthen the evidence of metabolic crosstalk between the heart and peripheral tissues through cardiokines and also set the stage for the development of novel treatments for metabolic syndrome.
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spelling doaj-art-cbc0a2c2d30e401b8e79c9b8224bb00f2025-08-20T04:02:56ZengSpringer NatureEMBO Molecular Medicine1757-46761757-46842014-12-016121521152410.15252/emmm.201404749Heart over mind: metabolic control of white adipose tissue and liverMichinari Nakamura0Junichi Sadoshima1Department of Cell Biology and Molecular Medicine, Cardiovascular Research Institute, Rutgers‐New Jersey Medical SchoolDepartment of Cell Biology and Molecular Medicine, Cardiovascular Research Institute, Rutgers‐New Jersey Medical SchoolAbstract Increasing evidence suggests that the heart controls the metabolism of peripheral organs. Olson and colleagues previously demonstrated that miR‐208a controls systemic energy homeostasis through the regulation of MED13 in cardiomyocytes (Grueter et al, 2012). In their follow‐up study in this issue of EMBO Molecular Medicine, white adipose tissue (WAT) and liver are identified as the physiological targets of cardiac MED13 signaling, most likely through cardiac‐derived circulating factors, which boost energy consumption by upregulating metabolic gene expression and increasing mitochondrial numbers (Baskin et al, 2014). In turn, increased energy expenditure in WAT and the liver confers leanness. These findings strengthen the evidence of metabolic crosstalk between the heart and peripheral tissues through cardiokines and also set the stage for the development of novel treatments for metabolic syndrome.https://doi.org/10.15252/emmm.201404749
spellingShingle Michinari Nakamura
Junichi Sadoshima
Heart over mind: metabolic control of white adipose tissue and liver
EMBO Molecular Medicine
title Heart over mind: metabolic control of white adipose tissue and liver
title_full Heart over mind: metabolic control of white adipose tissue and liver
title_fullStr Heart over mind: metabolic control of white adipose tissue and liver
title_full_unstemmed Heart over mind: metabolic control of white adipose tissue and liver
title_short Heart over mind: metabolic control of white adipose tissue and liver
title_sort heart over mind metabolic control of white adipose tissue and liver
url https://doi.org/10.15252/emmm.201404749
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