The decrease of GluN2B and its phosphorylation at Tyr-1336 in extrasynaptic subunits is associated with neuroprotection induced by hypoxia preconditioning

The decrease of GluN2B and its phosphorylation at Tyr-1336 in extrasynaptic subunits is associated with neuroprotection induced by hypoxia preconditioning

Prior research has firmly established that the N-methyl-d-aspartate (NMDA) receptor subunit 2 B (GluN2B) and its phosphorylation contribute to ischemic/hypoxic brain injury. Hypoxic preconditioning (HPC) is an endogenous mechanism that protects the brain from both ischaemic and hypoxic damage. In th...

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Bibliographic Details
Main Authors: Haidong Hou, Jing Yang, Gang Fu, Xiaolei Liu, You Liu, Kerui Gong, Ruifang Qi, Xiangning Jiang, Guo Shao
Format: Article
Language:English
Published: Elsevier 2025-07-01
Series:Brain Research Bulletin
Subjects:
Hypoxic preconditioning
Neuroprotection
GluN2B
Synapse
Online Access:http://www.sciencedirect.com/science/article/pii/S0361923025002126
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