The crosstalk between dendritic cells, cytokine-induced killer cells and cancer cells from the perspective of combination therapy

Dendritic cells (DCs) are considered the most potent professional antigen-presenting cells (APCs) that elicit adaptive antitumour immunity. DCs integrate multiple environmental signals by efficiently processing tumour-associated antigens (TAAs) and migrating to draining lymph nodes (dLNs), where the...

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Main Authors: Assel Issabekova, Marzhan Zhumabekova, Madina Zhunussova, Vyacheslav Ogay
Format: Article
Language:English
Published: Limited liability company «Science and Innovations» (Saratov) 2021-06-01
Series:Russian Open Medical Journal
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Online Access:https://romj.org/node/374
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author Assel Issabekova
Marzhan Zhumabekova
Madina Zhunussova
Vyacheslav Ogay
author_facet Assel Issabekova
Marzhan Zhumabekova
Madina Zhunussova
Vyacheslav Ogay
author_sort Assel Issabekova
collection DOAJ
description Dendritic cells (DCs) are considered the most potent professional antigen-presenting cells (APCs) that elicit adaptive antitumour immunity. DCs integrate multiple environmental signals by efficiently processing tumour-associated antigens (TAAs) and migrating to draining lymph nodes (dLNs), where they present foreign antigens to T cells for priming. DCs thus serve as a major link between innate and adaptive immunity. Although DCs (mostly monocyte-derived DCs [mo-DCs]) have already been used in cancer therapies, such approaches have shown limited efficacy. Mo-DCs have the unique ability to present antigens to T cells in peripheral tissues. CD3+CD56+ cytokine-induced killer (CIK) cells are characterized by both MHC-restricted and MHC-unrestricted antitumour cytotoxicity against a broad range of cancer cells. This review presents an overview of the mechanisms by which mo-DCs and CIK cells’ interact with each other and with tumours. The maturation of DCs was identified as a crucial step in the development of effective DC-based vaccines against cancer. A further improved adoptive immunotherapy strategy involves a combination of mature mo-DCs and CIK cells. Combination therapy presents many opportunities for cancer treatment, as reported by a number of clinical trials. However, there is a lack of fundamental studies on the interaction of in vitro-generated mo-DCs with CIK cells. We discuss several methods of boosting DC-based vaccines and review the current knowledge of contact-dependent and cytokine-induced interactions of mo-DCs with CIK cells. We highlight that the combination of mo-DCs with CIK cells activates MHC-restricted and MHC-unrestricted immune responses.
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spelling doaj-art-c736bcc77e3d42f1955f2d2e7249fb1b2025-08-20T03:07:33ZengLimited liability company «Science and Innovations» (Saratov)Russian Open Medical Journal2304-34152021-06-01102e020910.15275/rusomj.2021.0209The crosstalk between dendritic cells, cytokine-induced killer cells and cancer cells from the perspective of combination therapyAssel IssabekovaMarzhan ZhumabekovaMadina ZhunussovaVyacheslav OgayDendritic cells (DCs) are considered the most potent professional antigen-presenting cells (APCs) that elicit adaptive antitumour immunity. DCs integrate multiple environmental signals by efficiently processing tumour-associated antigens (TAAs) and migrating to draining lymph nodes (dLNs), where they present foreign antigens to T cells for priming. DCs thus serve as a major link between innate and adaptive immunity. Although DCs (mostly monocyte-derived DCs [mo-DCs]) have already been used in cancer therapies, such approaches have shown limited efficacy. Mo-DCs have the unique ability to present antigens to T cells in peripheral tissues. CD3+CD56+ cytokine-induced killer (CIK) cells are characterized by both MHC-restricted and MHC-unrestricted antitumour cytotoxicity against a broad range of cancer cells. This review presents an overview of the mechanisms by which mo-DCs and CIK cells’ interact with each other and with tumours. The maturation of DCs was identified as a crucial step in the development of effective DC-based vaccines against cancer. A further improved adoptive immunotherapy strategy involves a combination of mature mo-DCs and CIK cells. Combination therapy presents many opportunities for cancer treatment, as reported by a number of clinical trials. However, there is a lack of fundamental studies on the interaction of in vitro-generated mo-DCs with CIK cells. We discuss several methods of boosting DC-based vaccines and review the current knowledge of contact-dependent and cytokine-induced interactions of mo-DCs with CIK cells. We highlight that the combination of mo-DCs with CIK cells activates MHC-restricted and MHC-unrestricted immune responses.https://romj.org/node/374dendritic cellscytokine-induced killer cellsdendritic cells maturationheat-shock proteinsccr5 signalling
spellingShingle Assel Issabekova
Marzhan Zhumabekova
Madina Zhunussova
Vyacheslav Ogay
The crosstalk between dendritic cells, cytokine-induced killer cells and cancer cells from the perspective of combination therapy
Russian Open Medical Journal
dendritic cells
cytokine-induced killer cells
dendritic cells maturation
heat-shock proteins
ccr5 signalling
title The crosstalk between dendritic cells, cytokine-induced killer cells and cancer cells from the perspective of combination therapy
title_full The crosstalk between dendritic cells, cytokine-induced killer cells and cancer cells from the perspective of combination therapy
title_fullStr The crosstalk between dendritic cells, cytokine-induced killer cells and cancer cells from the perspective of combination therapy
title_full_unstemmed The crosstalk between dendritic cells, cytokine-induced killer cells and cancer cells from the perspective of combination therapy
title_short The crosstalk between dendritic cells, cytokine-induced killer cells and cancer cells from the perspective of combination therapy
title_sort crosstalk between dendritic cells cytokine induced killer cells and cancer cells from the perspective of combination therapy
topic dendritic cells
cytokine-induced killer cells
dendritic cells maturation
heat-shock proteins
ccr5 signalling
url https://romj.org/node/374
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