Mapping the Protein Phosphatase 1 Interactome in Human Cytomegalovirus Infection

Protein phosphorylation is a crucial regulatory mechanism in cellular homeostasis. The human cytomegalovirus (HCMV) incorporates protein phosphatase 1 (PP1) into its tegument, yet the biological relevance and mechanisms of this incorporation remain unclear. Our study offers the first characterizatio...

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Main Authors: Stefan Weinberger, Carmen Stecher, Marie-Theres Kastner, Sergei Nekhai, Christoph Steininger
Format: Article
Language:English
Published: MDPI AG 2024-12-01
Series:Viruses
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Online Access:https://www.mdpi.com/1999-4915/16/12/1961
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author Stefan Weinberger
Carmen Stecher
Marie-Theres Kastner
Sergei Nekhai
Christoph Steininger
author_facet Stefan Weinberger
Carmen Stecher
Marie-Theres Kastner
Sergei Nekhai
Christoph Steininger
author_sort Stefan Weinberger
collection DOAJ
description Protein phosphorylation is a crucial regulatory mechanism in cellular homeostasis. The human cytomegalovirus (HCMV) incorporates protein phosphatase 1 (PP1) into its tegument, yet the biological relevance and mechanisms of this incorporation remain unclear. Our study offers the first characterization of the PP1 interactome during HCMV infection and its alterations. Using co-immunoprecipitation, mass spectrometry, and quantitative proteomics, we identified 159 high-confidence interacting proteins (HCIPs) in the PP1 interactome, consisting of 126 human and 33 viral proteins. We observed significant temporal changes in the PP1 interactome following HCMV infection, including the altered interactions of PP1 regulatory subunits. Further analysis highlighted the central roles of these PP1 interacting proteins in intracellular trafficking, with particular emphasis on the trafficking protein particle complex and Rab GTPases, which are crucial for the virus’s manipulation of host cellular processes in virion assembly and egress. Additionally, our study on the noncatalytic PP1 inhibitor 1E7-03 revealed a decrease in PP1’s interaction with key HCMV proteins, supporting its potential as an antiviral agent. Our findings suggest that PP1 docking motifs are critical in viral–host interactions and offer new insights for antiviral strategies.
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spelling doaj-art-c4f658a8fc6d44cda7ab0d6bfb0731502024-12-27T14:59:22ZengMDPI AGViruses1999-49152024-12-011612196110.3390/v16121961Mapping the Protein Phosphatase 1 Interactome in Human Cytomegalovirus InfectionStefan Weinberger0Carmen Stecher1Marie-Theres Kastner2Sergei Nekhai3Christoph Steininger4Division of Infectious Diseases and Tropical Medicine, Department of Medicine I, Medical University of Vienna, 1090 Vienna, AustriaDivision of Infectious Diseases and Tropical Medicine, Department of Medicine I, Medical University of Vienna, 1090 Vienna, AustriaDivision of Infectious Diseases and Tropical Medicine, Department of Medicine I, Medical University of Vienna, 1090 Vienna, AustriaCenter for Sickle Cell Disease, Howard University, Washington, DC 20059, USADivision of Infectious Diseases and Tropical Medicine, Department of Medicine I, Medical University of Vienna, 1090 Vienna, AustriaProtein phosphorylation is a crucial regulatory mechanism in cellular homeostasis. The human cytomegalovirus (HCMV) incorporates protein phosphatase 1 (PP1) into its tegument, yet the biological relevance and mechanisms of this incorporation remain unclear. Our study offers the first characterization of the PP1 interactome during HCMV infection and its alterations. Using co-immunoprecipitation, mass spectrometry, and quantitative proteomics, we identified 159 high-confidence interacting proteins (HCIPs) in the PP1 interactome, consisting of 126 human and 33 viral proteins. We observed significant temporal changes in the PP1 interactome following HCMV infection, including the altered interactions of PP1 regulatory subunits. Further analysis highlighted the central roles of these PP1 interacting proteins in intracellular trafficking, with particular emphasis on the trafficking protein particle complex and Rab GTPases, which are crucial for the virus’s manipulation of host cellular processes in virion assembly and egress. Additionally, our study on the noncatalytic PP1 inhibitor 1E7-03 revealed a decrease in PP1’s interaction with key HCMV proteins, supporting its potential as an antiviral agent. Our findings suggest that PP1 docking motifs are critical in viral–host interactions and offer new insights for antiviral strategies.https://www.mdpi.com/1999-4915/16/12/1961cytomegalovirusprotein phosphatase 1interactomeproteomicsintracellular trafficking
spellingShingle Stefan Weinberger
Carmen Stecher
Marie-Theres Kastner
Sergei Nekhai
Christoph Steininger
Mapping the Protein Phosphatase 1 Interactome in Human Cytomegalovirus Infection
Viruses
cytomegalovirus
protein phosphatase 1
interactome
proteomics
intracellular trafficking
title Mapping the Protein Phosphatase 1 Interactome in Human Cytomegalovirus Infection
title_full Mapping the Protein Phosphatase 1 Interactome in Human Cytomegalovirus Infection
title_fullStr Mapping the Protein Phosphatase 1 Interactome in Human Cytomegalovirus Infection
title_full_unstemmed Mapping the Protein Phosphatase 1 Interactome in Human Cytomegalovirus Infection
title_short Mapping the Protein Phosphatase 1 Interactome in Human Cytomegalovirus Infection
title_sort mapping the protein phosphatase 1 interactome in human cytomegalovirus infection
topic cytomegalovirus
protein phosphatase 1
interactome
proteomics
intracellular trafficking
url https://www.mdpi.com/1999-4915/16/12/1961
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AT mariethereskastner mappingtheproteinphosphatase1interactomeinhumancytomegalovirusinfection
AT sergeinekhai mappingtheproteinphosphatase1interactomeinhumancytomegalovirusinfection
AT christophsteininger mappingtheproteinphosphatase1interactomeinhumancytomegalovirusinfection