Amyloid‐β oligomers suppress subunit‐specific glutamate receptor increase during LTP
Abstract Introduction Amyloid‐β oligomers (AβOs) are assumed to impair the ability of learning and memory by suppressing the induction of synaptic plasticity, such as long‐term potentiation (LTP) in the early stage of Alzheimer's disease. However, the direct molecular mechanism of how AβOs affe...
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| Main Authors: | Hiromitsu Tanaka, Daiki Sakaguchi, Tomoo Hirano |
|---|---|
| Format: | Article |
| Language: | English |
| Published: |
Wiley
2019-01-01
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| Series: | Alzheimer’s & Dementia: Translational Research & Clinical Interventions |
| Subjects: | |
| Online Access: | https://doi.org/10.1016/j.trci.2019.10.003 |
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