Crosstalk of pyroptosis and cytokine in the tumor microenvironment: from mechanisms to clinical implication

Abstract In the realm of cancer research, the tumor microenvironment (TME) plays a crucial role in tumor initiation and progression, shaped by complex interactions between cancer cells and surrounding non-cancerous cells. Cytokines, as essential immunomodulatory agents, are secreted by various cellu...

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Main Authors: Hua Wang, Tao Wang, Shuxiang Yan, Jinxin Tang, Yibo Zhang, Liming Wang, Haodong Xu, Chao Tu
Format: Article
Language:English
Published: BMC 2024-11-01
Series:Molecular Cancer
Subjects:
Online Access:https://doi.org/10.1186/s12943-024-02183-9
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author Hua Wang
Tao Wang
Shuxiang Yan
Jinxin Tang
Yibo Zhang
Liming Wang
Haodong Xu
Chao Tu
author_facet Hua Wang
Tao Wang
Shuxiang Yan
Jinxin Tang
Yibo Zhang
Liming Wang
Haodong Xu
Chao Tu
author_sort Hua Wang
collection DOAJ
description Abstract In the realm of cancer research, the tumor microenvironment (TME) plays a crucial role in tumor initiation and progression, shaped by complex interactions between cancer cells and surrounding non-cancerous cells. Cytokines, as essential immunomodulatory agents, are secreted by various cellular constituents within the TME, including immune cells, cancer-associated fibroblasts, and cancer cells themselves. These cytokines facilitate intricate communication networks that significantly influence tumor initiation, progression, metastasis, and immune suppression. Pyroptosis contributes to TME remodeling by promoting the release of pro-inflammatory cytokines and sustaining chronic inflammation, impacting processes such as immune escape and angiogenesis. However, challenges remain due to the complex interplay among cytokines, pyroptosis, and the TME, along with the dual effects of pyroptosis on cancer progression and therapy-related complications like cytokine release syndrome. Unraveling these complexities could facilitate strategies that balance inflammatory responses while minimizing tissue damage during therapy. This review delves into the complex crosstalk between cytokines, pyroptosis, and the TME, elucidating their contribution to tumor progression and metastasis. By synthesizing emerging therapeutic targets and innovative technologies concerning TME, this review aims to provide novel insights that could enhance treatment outcomes for cancer patients.
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series Molecular Cancer
spelling doaj-art-ad34a8c3945d48cfb7c464e89a09def52024-12-01T12:12:54ZengBMCMolecular Cancer1476-45982024-11-0123113710.1186/s12943-024-02183-9Crosstalk of pyroptosis and cytokine in the tumor microenvironment: from mechanisms to clinical implicationHua Wang0Tao Wang1Shuxiang Yan2Jinxin Tang3Yibo Zhang4Liming Wang5Haodong Xu6Chao Tu7Department of Orthopaedics, The Second Xiangya Hospital of Central South UniversityDepartment of Orthopaedics, The Second Xiangya Hospital of Central South UniversityDepartment of Nephrology, The Second Xiangya Hospital of Central South UniversityDepartment of Orthopaedics, The Second Xiangya Hospital of Central South UniversityDepartment of Orthopaedics, The Second Xiangya Hospital of Central South UniversitySchool of Biomedical Sciences, Hunan UniversityDepartment of Orthopaedics, The Second Xiangya Hospital of Central South UniversityDepartment of Orthopaedics, The Second Xiangya Hospital of Central South UniversityAbstract In the realm of cancer research, the tumor microenvironment (TME) plays a crucial role in tumor initiation and progression, shaped by complex interactions between cancer cells and surrounding non-cancerous cells. Cytokines, as essential immunomodulatory agents, are secreted by various cellular constituents within the TME, including immune cells, cancer-associated fibroblasts, and cancer cells themselves. These cytokines facilitate intricate communication networks that significantly influence tumor initiation, progression, metastasis, and immune suppression. Pyroptosis contributes to TME remodeling by promoting the release of pro-inflammatory cytokines and sustaining chronic inflammation, impacting processes such as immune escape and angiogenesis. However, challenges remain due to the complex interplay among cytokines, pyroptosis, and the TME, along with the dual effects of pyroptosis on cancer progression and therapy-related complications like cytokine release syndrome. Unraveling these complexities could facilitate strategies that balance inflammatory responses while minimizing tissue damage during therapy. This review delves into the complex crosstalk between cytokines, pyroptosis, and the TME, elucidating their contribution to tumor progression and metastasis. By synthesizing emerging therapeutic targets and innovative technologies concerning TME, this review aims to provide novel insights that could enhance treatment outcomes for cancer patients.https://doi.org/10.1186/s12943-024-02183-9PyroptosisCytokineTumor microenvironmentMechanismClinical implication
spellingShingle Hua Wang
Tao Wang
Shuxiang Yan
Jinxin Tang
Yibo Zhang
Liming Wang
Haodong Xu
Chao Tu
Crosstalk of pyroptosis and cytokine in the tumor microenvironment: from mechanisms to clinical implication
Molecular Cancer
Pyroptosis
Cytokine
Tumor microenvironment
Mechanism
Clinical implication
title Crosstalk of pyroptosis and cytokine in the tumor microenvironment: from mechanisms to clinical implication
title_full Crosstalk of pyroptosis and cytokine in the tumor microenvironment: from mechanisms to clinical implication
title_fullStr Crosstalk of pyroptosis and cytokine in the tumor microenvironment: from mechanisms to clinical implication
title_full_unstemmed Crosstalk of pyroptosis and cytokine in the tumor microenvironment: from mechanisms to clinical implication
title_short Crosstalk of pyroptosis and cytokine in the tumor microenvironment: from mechanisms to clinical implication
title_sort crosstalk of pyroptosis and cytokine in the tumor microenvironment from mechanisms to clinical implication
topic Pyroptosis
Cytokine
Tumor microenvironment
Mechanism
Clinical implication
url https://doi.org/10.1186/s12943-024-02183-9
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