Silver-quercetin-loaded honeycomb-like Ti-based interface combats infection-triggered excessive inflammation via specific bactericidal and macrophage reprogramming

Excessive inflammation caused by bacterial infection is the primary cause of implant failure. Antibiotic treatment often fails to prevent peri-implant infection and may induce unexpected drug resistance. Herein, a non-antibiotic strategy based on the synergy of silver ion release and macrophage repr...

Full description

Saved in:
Bibliographic Details
Main Authors: Ning Yang, Ting Wu, Meng Li, Xianli Hu, Ruixiang Ma, Wei Jiang, Zheng Su, Rong Yang, Chen Zhu
Format: Article
Language:English
Published: KeAi Communications Co., Ltd. 2025-01-01
Series:Bioactive Materials
Subjects:
Online Access:http://www.sciencedirect.com/science/article/pii/S2452199X24003992
Tags: Add Tag
No Tags, Be the first to tag this record!
_version_ 1846157826956722176
author Ning Yang
Ting Wu
Meng Li
Xianli Hu
Ruixiang Ma
Wei Jiang
Zheng Su
Rong Yang
Chen Zhu
author_facet Ning Yang
Ting Wu
Meng Li
Xianli Hu
Ruixiang Ma
Wei Jiang
Zheng Su
Rong Yang
Chen Zhu
author_sort Ning Yang
collection DOAJ
description Excessive inflammation caused by bacterial infection is the primary cause of implant failure. Antibiotic treatment often fails to prevent peri-implant infection and may induce unexpected drug resistance. Herein, a non-antibiotic strategy based on the synergy of silver ion release and macrophage reprogramming is proposed for preventing infection and bacteria-induced inflammation suppression by the organic-inorganic hybridization of silver nanoparticle (AgNP) and quercetin (Que) into a polydopamine (PDA)-based coating on the 3D framework of porous titanium (SQPdFT). Once the planktonic bacteria (e.g., Escherichia coli, Staphylococcus aureus) reach the surface of SQPdFT, released Que disrupts the bacterial membrane. Then, AgNP can penetrate the invading bacterium and kill them, which further inhibits the biofilm formation. Simultaneously, released Que can regulate macrophage polarization homeostasis via the peroxisome proliferators-activated receptors gamma (PPARγ)-mediated nuclear factor kappa-B (NF-κB) pathway, thereby terminating excessive inflammatory responses. These advantages facilitate the adhesion and osteogenic differentiation of bone marrow-derived mesenchymal stem cells (BMSCs), concomitantly suppressing osteoclast maturation, and eventually conferring superior mechanical stability to SQPdFT within the medullary cavity. In summary, owing to its excellent antibacterial effect, immune remodeling function, and pro-osteointegration ability, SQPdFT is a promising protective coating for titanium-based implants used in orthopedic replacement surgery.
format Article
id doaj-art-8e9a9c2b4ae44f2c97d91afb4154b9f4
institution Kabale University
issn 2452-199X
language English
publishDate 2025-01-01
publisher KeAi Communications Co., Ltd.
record_format Article
series Bioactive Materials
spelling doaj-art-8e9a9c2b4ae44f2c97d91afb4154b9f42024-11-25T04:41:32ZengKeAi Communications Co., Ltd.Bioactive Materials2452-199X2025-01-01434866Silver-quercetin-loaded honeycomb-like Ti-based interface combats infection-triggered excessive inflammation via specific bactericidal and macrophage reprogrammingNing Yang0Ting Wu1Meng Li2Xianli Hu3Ruixiang Ma4Wei Jiang5Zheng Su6Rong Yang7Chen Zhu8Department of Orthopaedics, Centre for Leading Medicine and Advanced Technologies of IHM, The First Affiliated Hospital of USTC, Division of Life Sciences and Medicine, University of Science and Technology of China, Hefei, 230001, Anhui, ChinaCAS Key Lab for Biomedical Effects of Nanomaterials and Nanosafety, Center of Materials Science and Optoelectronics Engineering, National Center for Nanoscience and Technology, University of Chinese Academy of Sciences, Beijing, 100190, ChinaDepartment of Orthopaedics, Centre for Leading Medicine and Advanced Technologies of IHM, The First Affiliated Hospital of USTC, Division of Life Sciences and Medicine, University of Science and Technology of China, Hefei, 230001, Anhui, ChinaDepartment of Orthopaedics, Centre for Leading Medicine and Advanced Technologies of IHM, The First Affiliated Hospital of USTC, Division of Life Sciences and Medicine, University of Science and Technology of China, Hefei, 230001, Anhui, ChinaDepartment of Orthopaedics, Centre for Leading Medicine and Advanced Technologies of IHM, The First Affiliated Hospital of USTC, Division of Life Sciences and Medicine, University of Science and Technology of China, Hefei, 230001, Anhui, ChinaDepartment of Orthopedics, The First Affiliated Hospital of Anhui Medical University, Hefei, Anhui, 230022, ChinaDepartment of Orthopaedics, Centre for Leading Medicine and Advanced Technologies of IHM, The First Affiliated Hospital of USTC, Division of Life Sciences and Medicine, University of Science and Technology of China, Hefei, 230001, Anhui, China; Corresponding author.CAS Key Lab for Biomedical Effects of Nanomaterials and Nanosafety, Center of Materials Science and Optoelectronics Engineering, National Center for Nanoscience and Technology, University of Chinese Academy of Sciences, Beijing, 100190, China; Corresponding author.Department of Orthopaedics, Centre for Leading Medicine and Advanced Technologies of IHM, The First Affiliated Hospital of USTC, Division of Life Sciences and Medicine, University of Science and Technology of China, Hefei, 230001, Anhui, China; Corresponding author.Excessive inflammation caused by bacterial infection is the primary cause of implant failure. Antibiotic treatment often fails to prevent peri-implant infection and may induce unexpected drug resistance. Herein, a non-antibiotic strategy based on the synergy of silver ion release and macrophage reprogramming is proposed for preventing infection and bacteria-induced inflammation suppression by the organic-inorganic hybridization of silver nanoparticle (AgNP) and quercetin (Que) into a polydopamine (PDA)-based coating on the 3D framework of porous titanium (SQPdFT). Once the planktonic bacteria (e.g., Escherichia coli, Staphylococcus aureus) reach the surface of SQPdFT, released Que disrupts the bacterial membrane. Then, AgNP can penetrate the invading bacterium and kill them, which further inhibits the biofilm formation. Simultaneously, released Que can regulate macrophage polarization homeostasis via the peroxisome proliferators-activated receptors gamma (PPARγ)-mediated nuclear factor kappa-B (NF-κB) pathway, thereby terminating excessive inflammatory responses. These advantages facilitate the adhesion and osteogenic differentiation of bone marrow-derived mesenchymal stem cells (BMSCs), concomitantly suppressing osteoclast maturation, and eventually conferring superior mechanical stability to SQPdFT within the medullary cavity. In summary, owing to its excellent antibacterial effect, immune remodeling function, and pro-osteointegration ability, SQPdFT is a promising protective coating for titanium-based implants used in orthopedic replacement surgery.http://www.sciencedirect.com/science/article/pii/S2452199X24003992Silver nanoparticlesQuercetinPorous titaniumMacrophage polarization homeostasisOsteointegration
spellingShingle Ning Yang
Ting Wu
Meng Li
Xianli Hu
Ruixiang Ma
Wei Jiang
Zheng Su
Rong Yang
Chen Zhu
Silver-quercetin-loaded honeycomb-like Ti-based interface combats infection-triggered excessive inflammation via specific bactericidal and macrophage reprogramming
Bioactive Materials
Silver nanoparticles
Quercetin
Porous titanium
Macrophage polarization homeostasis
Osteointegration
title Silver-quercetin-loaded honeycomb-like Ti-based interface combats infection-triggered excessive inflammation via specific bactericidal and macrophage reprogramming
title_full Silver-quercetin-loaded honeycomb-like Ti-based interface combats infection-triggered excessive inflammation via specific bactericidal and macrophage reprogramming
title_fullStr Silver-quercetin-loaded honeycomb-like Ti-based interface combats infection-triggered excessive inflammation via specific bactericidal and macrophage reprogramming
title_full_unstemmed Silver-quercetin-loaded honeycomb-like Ti-based interface combats infection-triggered excessive inflammation via specific bactericidal and macrophage reprogramming
title_short Silver-quercetin-loaded honeycomb-like Ti-based interface combats infection-triggered excessive inflammation via specific bactericidal and macrophage reprogramming
title_sort silver quercetin loaded honeycomb like ti based interface combats infection triggered excessive inflammation via specific bactericidal and macrophage reprogramming
topic Silver nanoparticles
Quercetin
Porous titanium
Macrophage polarization homeostasis
Osteointegration
url http://www.sciencedirect.com/science/article/pii/S2452199X24003992
work_keys_str_mv AT ningyang silverquercetinloadedhoneycombliketibasedinterfacecombatsinfectiontriggeredexcessiveinflammationviaspecificbactericidalandmacrophagereprogramming
AT tingwu silverquercetinloadedhoneycombliketibasedinterfacecombatsinfectiontriggeredexcessiveinflammationviaspecificbactericidalandmacrophagereprogramming
AT mengli silverquercetinloadedhoneycombliketibasedinterfacecombatsinfectiontriggeredexcessiveinflammationviaspecificbactericidalandmacrophagereprogramming
AT xianlihu silverquercetinloadedhoneycombliketibasedinterfacecombatsinfectiontriggeredexcessiveinflammationviaspecificbactericidalandmacrophagereprogramming
AT ruixiangma silverquercetinloadedhoneycombliketibasedinterfacecombatsinfectiontriggeredexcessiveinflammationviaspecificbactericidalandmacrophagereprogramming
AT weijiang silverquercetinloadedhoneycombliketibasedinterfacecombatsinfectiontriggeredexcessiveinflammationviaspecificbactericidalandmacrophagereprogramming
AT zhengsu silverquercetinloadedhoneycombliketibasedinterfacecombatsinfectiontriggeredexcessiveinflammationviaspecificbactericidalandmacrophagereprogramming
AT rongyang silverquercetinloadedhoneycombliketibasedinterfacecombatsinfectiontriggeredexcessiveinflammationviaspecificbactericidalandmacrophagereprogramming
AT chenzhu silverquercetinloadedhoneycombliketibasedinterfacecombatsinfectiontriggeredexcessiveinflammationviaspecificbactericidalandmacrophagereprogramming