Autophagy deficiency protects against ocular hypertension and glaucoma

Glaucoma encompasses a spectrum of disorders characterized by the chronic degeneration of retinal ganglion cell (RGC) axons and the progressive loss of RGCs, resulting in visual impairment. In this study, we investigated the effect of autophagy deficiency on two glaucoma hypertensive models, the DBA...

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Main Authors: Angela Dixon, Myoung Sup Shim, April Nettesheim, Aislyn Coyne, Chien-Chia Su, Haiyan Gong, Paloma B. Liton
Format: Article
Language:English
Published: Taylor & Francis Group 2024-12-01
Series:Autophagy Reports
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Online Access:https://www.tandfonline.com/doi/10.1080/27694127.2023.2285214
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author Angela Dixon
Myoung Sup Shim
April Nettesheim
Aislyn Coyne
Chien-Chia Su
Haiyan Gong
Paloma B. Liton
author_facet Angela Dixon
Myoung Sup Shim
April Nettesheim
Aislyn Coyne
Chien-Chia Su
Haiyan Gong
Paloma B. Liton
author_sort Angela Dixon
collection DOAJ
description Glaucoma encompasses a spectrum of disorders characterized by the chronic degeneration of retinal ganglion cell (RGC) axons and the progressive loss of RGCs, resulting in visual impairment. In this study, we investigated the effect of autophagy deficiency on two glaucoma hypertensive models, the DBA/2J spontaneous glaucoma model, and the TGFβ2 (transforming growth factor β2) chronic ocular hypertensive model. For this, we used the Atg4b−/− and DBA/2J-Atg4b−/− mice, this latter generated in our laboratory via CRISPR/Cas9 technology, which display impaired autophagy. In contrast to littermate WT controls, mice deficient in Atg4B, did not develop glaucomatous elevation of intraocular pressure (IOP). Moreover, autophagy deficiency protected against RGC death and optic nerve atrophy. Collectively, our data supports a pathogenic role of autophagy in the context of ocular hypertension and glaucoma.Abbreviations: ATG4B: Autophagy-related 4B; BAX: BCL2-associated X protein; BECN1: Beclin 1; BID: BH3 interacting domain death agonist; CASP8: Caspase 8; IOP: Intraocular Pressure; MAP1LC3B: microtubule-associated proteins 1B; ON: Optic Nerve; RGC: Retinal Ganglion Cells; SQSTSM1: Sequestosome 1; TBK1: TANK-binding kinase 1; TGFβ2: Transforming Growth Factor β2; WT: Wild Type.
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spelling doaj-art-868fae2bf19d49f1a28a82eb56a4edb12024-12-09T07:19:32ZengTaylor & Francis GroupAutophagy Reports2769-41272024-12-013110.1080/27694127.2023.2285214Autophagy deficiency protects against ocular hypertension and glaucomaAngela Dixon0Myoung Sup Shim1April Nettesheim2Aislyn Coyne3Chien-Chia Su4Haiyan Gong5Paloma B. Liton6Department of Ophthalmology & Pathology, Duke University, Durham, NC, 27705, USADepartment of Ophthalmology & Pathology, Duke University, Durham, NC, 27705, USADepartment of Ophthalmology & Pathology, Duke University, Durham, NC, 27705, USADepartment of Ophthalmology & Pathology, Duke University, Durham, NC, 27705, USADepartment of Ophthalmology & Pathology, Duke University, Durham, NC, 27705, USADepartment of Ophthalmology, Boston University School of Medicine, Boston, MA, 02118, USADepartment of Ophthalmology & Pathology, Duke University, Durham, NC, 27705, USAGlaucoma encompasses a spectrum of disorders characterized by the chronic degeneration of retinal ganglion cell (RGC) axons and the progressive loss of RGCs, resulting in visual impairment. In this study, we investigated the effect of autophagy deficiency on two glaucoma hypertensive models, the DBA/2J spontaneous glaucoma model, and the TGFβ2 (transforming growth factor β2) chronic ocular hypertensive model. For this, we used the Atg4b−/− and DBA/2J-Atg4b−/− mice, this latter generated in our laboratory via CRISPR/Cas9 technology, which display impaired autophagy. In contrast to littermate WT controls, mice deficient in Atg4B, did not develop glaucomatous elevation of intraocular pressure (IOP). Moreover, autophagy deficiency protected against RGC death and optic nerve atrophy. Collectively, our data supports a pathogenic role of autophagy in the context of ocular hypertension and glaucoma.Abbreviations: ATG4B: Autophagy-related 4B; BAX: BCL2-associated X protein; BECN1: Beclin 1; BID: BH3 interacting domain death agonist; CASP8: Caspase 8; IOP: Intraocular Pressure; MAP1LC3B: microtubule-associated proteins 1B; ON: Optic Nerve; RGC: Retinal Ganglion Cells; SQSTSM1: Sequestosome 1; TBK1: TANK-binding kinase 1; TGFβ2: Transforming Growth Factor β2; WT: Wild Type.https://www.tandfonline.com/doi/10.1080/27694127.2023.2285214Autophagyocular hypertensionglaucomaRGCoptic nerveDBA/2J
spellingShingle Angela Dixon
Myoung Sup Shim
April Nettesheim
Aislyn Coyne
Chien-Chia Su
Haiyan Gong
Paloma B. Liton
Autophagy deficiency protects against ocular hypertension and glaucoma
Autophagy Reports
Autophagy
ocular hypertension
glaucoma
RGC
optic nerve
DBA/2J
title Autophagy deficiency protects against ocular hypertension and glaucoma
title_full Autophagy deficiency protects against ocular hypertension and glaucoma
title_fullStr Autophagy deficiency protects against ocular hypertension and glaucoma
title_full_unstemmed Autophagy deficiency protects against ocular hypertension and glaucoma
title_short Autophagy deficiency protects against ocular hypertension and glaucoma
title_sort autophagy deficiency protects against ocular hypertension and glaucoma
topic Autophagy
ocular hypertension
glaucoma
RGC
optic nerve
DBA/2J
url https://www.tandfonline.com/doi/10.1080/27694127.2023.2285214
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