Activation of BDNF–TrkB Signaling in Specific Structures of the Sheep Brain by Kynurenic Acid

Fluctuations in kynurenic acid (KYNA) and brain-derived neurotrophic factor (BDNF) levels in the brain reflect its neurological status. The aim of the study was to investigate the effect of transiently elevated KYNA concentrations in the cerebroventricular circulation on the expression of BDNF and i...

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Main Authors: Katarzyna Roszkowicz-Ostrowska, Patrycja Młotkowska, Elżbieta Marciniak, Michał Szlis, Marcin Barszcz, Tomasz Misztal
Format: Article
Language:English
Published: MDPI AG 2024-11-01
Series:Cells
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Online Access:https://www.mdpi.com/2073-4409/13/23/1928
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author Katarzyna Roszkowicz-Ostrowska
Patrycja Młotkowska
Elżbieta Marciniak
Michał Szlis
Marcin Barszcz
Tomasz Misztal
author_facet Katarzyna Roszkowicz-Ostrowska
Patrycja Młotkowska
Elżbieta Marciniak
Michał Szlis
Marcin Barszcz
Tomasz Misztal
author_sort Katarzyna Roszkowicz-Ostrowska
collection DOAJ
description Fluctuations in kynurenic acid (KYNA) and brain-derived neurotrophic factor (BDNF) levels in the brain reflect its neurological status. The aim of the study was to investigate the effect of transiently elevated KYNA concentrations in the cerebroventricular circulation on the expression of BDNF and its high-affinity tropomyosin-related kinase receptor B (TrkB) in specific structures of the sheep brain. Intracerebroventricularly cannulated anestrous sheep were subjected to a series of four 30 min infusions of KYNA: 4 × 5 μg/60 μL/30 min (KYNA20, <i>n</i> = 6) and 4 × 25 μg/60 μL/30 min (KYNA100, <i>n</i> = 6) or a control infusion (<i>n</i> = 6), at 30 min intervals. Sections of the hippocampal CA3 field, amygdala (AMG), prefrontal cortex (PCx), and the hypothalamic medial-basal (MBH) and preoptic (POA) areas were dissected from the brain immediately after the experiment. The highest concentration of BDNF protein was found in the CA3 field (<i>p</i> < 0.001), which was 8-fold higher than in the AMG and 12-fold higher than that in the PCx (MBH and POA were not analyzed). The most pronounced BDNF mRNA expression was observed in the MBH, followed by the PCx, POA, AMG and CA3, while the highest abundance of TrkB mRNA was recorded in the AMG, followed by the MBH, PCx, CA3, and POA. KYNA increased (<i>p</i> < 0.05–<i>p</i> < 0.01) BDNF protein levels and the expression of its gene in the brain structures were examined, with the effect varying by dose and brain region. KYNA, particularly at the KYNA100 dose, also increased (<i>p</i> < 0.01) <i>TrkB</i> gene expression, except for the AMG, where the lower KYNA20 dose was more effective (<i>p</i> < 0.01). These findings suggest a positive relationship between KYNA levels in the cerebroventricular circulation and BDNF–TrkB expression in specific brain regions in a sheep model. This indicates that a transient increase in the CSF KYNA concentration can potentially restore BDNF production, for which deficiency underlies numerous neurological disorders.
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spelling doaj-art-827ca9c9e9ff4b63a6cd729b2786ad0d2024-12-13T16:24:02ZengMDPI AGCells2073-44092024-11-011323192810.3390/cells13231928Activation of BDNF–TrkB Signaling in Specific Structures of the Sheep Brain by Kynurenic AcidKatarzyna Roszkowicz-Ostrowska0Patrycja Młotkowska1Elżbieta Marciniak2Michał Szlis3Marcin Barszcz4Tomasz Misztal5The Kielanowski Institute of Animal Physiology and Nutrition, Polish Academy of Sciences, Instytucka 3 Str., 05-110 Jabłonna, PolandThe Kielanowski Institute of Animal Physiology and Nutrition, Polish Academy of Sciences, Instytucka 3 Str., 05-110 Jabłonna, PolandThe Kielanowski Institute of Animal Physiology and Nutrition, Polish Academy of Sciences, Instytucka 3 Str., 05-110 Jabłonna, PolandThe Kielanowski Institute of Animal Physiology and Nutrition, Polish Academy of Sciences, Instytucka 3 Str., 05-110 Jabłonna, PolandThe Kielanowski Institute of Animal Physiology and Nutrition, Polish Academy of Sciences, Instytucka 3 Str., 05-110 Jabłonna, PolandThe Kielanowski Institute of Animal Physiology and Nutrition, Polish Academy of Sciences, Instytucka 3 Str., 05-110 Jabłonna, PolandFluctuations in kynurenic acid (KYNA) and brain-derived neurotrophic factor (BDNF) levels in the brain reflect its neurological status. The aim of the study was to investigate the effect of transiently elevated KYNA concentrations in the cerebroventricular circulation on the expression of BDNF and its high-affinity tropomyosin-related kinase receptor B (TrkB) in specific structures of the sheep brain. Intracerebroventricularly cannulated anestrous sheep were subjected to a series of four 30 min infusions of KYNA: 4 × 5 μg/60 μL/30 min (KYNA20, <i>n</i> = 6) and 4 × 25 μg/60 μL/30 min (KYNA100, <i>n</i> = 6) or a control infusion (<i>n</i> = 6), at 30 min intervals. Sections of the hippocampal CA3 field, amygdala (AMG), prefrontal cortex (PCx), and the hypothalamic medial-basal (MBH) and preoptic (POA) areas were dissected from the brain immediately after the experiment. The highest concentration of BDNF protein was found in the CA3 field (<i>p</i> < 0.001), which was 8-fold higher than in the AMG and 12-fold higher than that in the PCx (MBH and POA were not analyzed). The most pronounced BDNF mRNA expression was observed in the MBH, followed by the PCx, POA, AMG and CA3, while the highest abundance of TrkB mRNA was recorded in the AMG, followed by the MBH, PCx, CA3, and POA. KYNA increased (<i>p</i> < 0.05–<i>p</i> < 0.01) BDNF protein levels and the expression of its gene in the brain structures were examined, with the effect varying by dose and brain region. KYNA, particularly at the KYNA100 dose, also increased (<i>p</i> < 0.01) <i>TrkB</i> gene expression, except for the AMG, where the lower KYNA20 dose was more effective (<i>p</i> < 0.01). These findings suggest a positive relationship between KYNA levels in the cerebroventricular circulation and BDNF–TrkB expression in specific brain regions in a sheep model. This indicates that a transient increase in the CSF KYNA concentration can potentially restore BDNF production, for which deficiency underlies numerous neurological disorders.https://www.mdpi.com/2073-4409/13/23/1928kynurenic acidintracerebroventricular infusionBDNFTrkBsheep brain
spellingShingle Katarzyna Roszkowicz-Ostrowska
Patrycja Młotkowska
Elżbieta Marciniak
Michał Szlis
Marcin Barszcz
Tomasz Misztal
Activation of BDNF–TrkB Signaling in Specific Structures of the Sheep Brain by Kynurenic Acid
Cells
kynurenic acid
intracerebroventricular infusion
BDNF
TrkB
sheep brain
title Activation of BDNF–TrkB Signaling in Specific Structures of the Sheep Brain by Kynurenic Acid
title_full Activation of BDNF–TrkB Signaling in Specific Structures of the Sheep Brain by Kynurenic Acid
title_fullStr Activation of BDNF–TrkB Signaling in Specific Structures of the Sheep Brain by Kynurenic Acid
title_full_unstemmed Activation of BDNF–TrkB Signaling in Specific Structures of the Sheep Brain by Kynurenic Acid
title_short Activation of BDNF–TrkB Signaling in Specific Structures of the Sheep Brain by Kynurenic Acid
title_sort activation of bdnf trkb signaling in specific structures of the sheep brain by kynurenic acid
topic kynurenic acid
intracerebroventricular infusion
BDNF
TrkB
sheep brain
url https://www.mdpi.com/2073-4409/13/23/1928
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AT elzbietamarciniak activationofbdnftrkbsignalinginspecificstructuresofthesheepbrainbykynurenicacid
AT michałszlis activationofbdnftrkbsignalinginspecificstructuresofthesheepbrainbykynurenicacid
AT marcinbarszcz activationofbdnftrkbsignalinginspecificstructuresofthesheepbrainbykynurenicacid
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