P4HA2 promotes tumor progression and is transcriptionally regulated by SP1 in colorectal cancer

P4HA2 has been implicated in various malignant tumors; however, its expression and functional role in colorectal cancer (CRC) remain poorly elucidated. This study aims to investigate the involvement of P4HA2 in CRC metastasis and progression, uncovering the underlying mechanisms. In colorectal cance...

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Main Authors: Xuening Dang, Xiaojian Chen, Zhonglin Liang, Zhujiang Dai, Wenjun Ding, Jinglue Song, Jihong Fu
Format: Article
Language:English
Published: Taylor & Francis Group 2024-12-01
Series:Cancer Biology & Therapy
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Online Access:https://www.tandfonline.com/doi/10.1080/15384047.2024.2361594
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author Xuening Dang
Xiaojian Chen
Zhonglin Liang
Zhujiang Dai
Wenjun Ding
Jinglue Song
Jihong Fu
author_facet Xuening Dang
Xiaojian Chen
Zhonglin Liang
Zhujiang Dai
Wenjun Ding
Jinglue Song
Jihong Fu
author_sort Xuening Dang
collection DOAJ
description P4HA2 has been implicated in various malignant tumors; however, its expression and functional role in colorectal cancer (CRC) remain poorly elucidated. This study aims to investigate the involvement of P4HA2 in CRC metastasis and progression, uncovering the underlying mechanisms. In colorectal cancer (CRC), P4HA2 exhibited overexpression, and elevated levels of P4HA2 expression were associated with an unfavorable prognosis. Functional assays demonstrated P4HA2‘s regulation of cell proliferation, and epithelial–mesenchymal transition (EMT) both in vitro and in vivo. Additionally, the AGO1 expression was correlated with P4HA2, and depletion of AGO1 reversed the proliferation and EMT function induced by P4HA2. Chromatin immunoprecipitation (ChIP) and luciferase assays suggested that the transcription factor SP1 binds to the promoter sequence of P4HA2, activating its expression in CRC. This study unveiled SP1 as a transcriptional regulator of P4HA2 in CRC and AGO1 is a probable target of P4HA2. In conclusion, P4HA2 emerges as a potential prognostic biomarker and promising therapeutic target in colorectal cancer.
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issn 1538-4047
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language English
publishDate 2024-12-01
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series Cancer Biology & Therapy
spelling doaj-art-8129cea262d1486e8f5b99db112a6dfe2024-12-13T06:53:25ZengTaylor & Francis GroupCancer Biology & Therapy1538-40471555-85762024-12-0125110.1080/15384047.2024.2361594P4HA2 promotes tumor progression and is transcriptionally regulated by SP1 in colorectal cancerXuening Dang0Xiaojian Chen1Zhonglin Liang2Zhujiang Dai3Wenjun Ding4Jinglue Song5Jihong Fu6Department of Colorectal and Anal Surgery, Xinhua Hospital, Shanghai Jiaotong University School of Medicine, Shanghai, ChinaDepartment of Colorectal and Anal Surgery, Xinhua Hospital, Shanghai Jiaotong University School of Medicine, Shanghai, ChinaDepartment of Colorectal and Anal Surgery, Xinhua Hospital, Shanghai Jiaotong University School of Medicine, Shanghai, ChinaDepartment of Colorectal and Anal Surgery, Xinhua Hospital, Shanghai Jiaotong University School of Medicine, Shanghai, ChinaDepartment of Colorectal and Anal Surgery, Xinhua Hospital, Shanghai Jiaotong University School of Medicine, Shanghai, ChinaDepartment of Colorectal and Anal Surgery, Xinhua Hospital, Shanghai Jiaotong University School of Medicine, Shanghai, ChinaDepartment of Colorectal and Anal Surgery, Xinhua Hospital, Shanghai Jiaotong University School of Medicine, Shanghai, ChinaP4HA2 has been implicated in various malignant tumors; however, its expression and functional role in colorectal cancer (CRC) remain poorly elucidated. This study aims to investigate the involvement of P4HA2 in CRC metastasis and progression, uncovering the underlying mechanisms. In colorectal cancer (CRC), P4HA2 exhibited overexpression, and elevated levels of P4HA2 expression were associated with an unfavorable prognosis. Functional assays demonstrated P4HA2‘s regulation of cell proliferation, and epithelial–mesenchymal transition (EMT) both in vitro and in vivo. Additionally, the AGO1 expression was correlated with P4HA2, and depletion of AGO1 reversed the proliferation and EMT function induced by P4HA2. Chromatin immunoprecipitation (ChIP) and luciferase assays suggested that the transcription factor SP1 binds to the promoter sequence of P4HA2, activating its expression in CRC. This study unveiled SP1 as a transcriptional regulator of P4HA2 in CRC and AGO1 is a probable target of P4HA2. In conclusion, P4HA2 emerges as a potential prognostic biomarker and promising therapeutic target in colorectal cancer.https://www.tandfonline.com/doi/10.1080/15384047.2024.2361594P4HA2colorectal cancerEMTAGO1SP1
spellingShingle Xuening Dang
Xiaojian Chen
Zhonglin Liang
Zhujiang Dai
Wenjun Ding
Jinglue Song
Jihong Fu
P4HA2 promotes tumor progression and is transcriptionally regulated by SP1 in colorectal cancer
Cancer Biology & Therapy
P4HA2
colorectal cancer
EMT
AGO1
SP1
title P4HA2 promotes tumor progression and is transcriptionally regulated by SP1 in colorectal cancer
title_full P4HA2 promotes tumor progression and is transcriptionally regulated by SP1 in colorectal cancer
title_fullStr P4HA2 promotes tumor progression and is transcriptionally regulated by SP1 in colorectal cancer
title_full_unstemmed P4HA2 promotes tumor progression and is transcriptionally regulated by SP1 in colorectal cancer
title_short P4HA2 promotes tumor progression and is transcriptionally regulated by SP1 in colorectal cancer
title_sort p4ha2 promotes tumor progression and is transcriptionally regulated by sp1 in colorectal cancer
topic P4HA2
colorectal cancer
EMT
AGO1
SP1
url https://www.tandfonline.com/doi/10.1080/15384047.2024.2361594
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