A new hERG allosteric modulator rescues genetic and drug‐induced long‐QT syndrome phenotypes in cardiomyocytes from isogenic pairs of patient induced pluripotent stem cells

Abstract Long‐QT syndrome (LQTS) is an arrhythmogenic disorder characterised by prolongation of the QT interval in the electrocardiogram, which can lead to sudden cardiac death. Pharmacological treatments are far from optimal for congenital forms of LQTS, while the acquired form, often triggered by...

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Main Authors: Luca Sala, Zhiyi Yu, Dorien Ward‐van Oostwaard, Jacobus PD van Veldhoven, Alessandra Moretti, Karl‐Ludwig Laugwitz, Christine L Mummery, Adriaan P IJzerman, Milena Bellin
Format: Article
Language:English
Published: Springer Nature 2016-07-01
Series:EMBO Molecular Medicine
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Online Access:https://doi.org/10.15252/emmm.201606260
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author Luca Sala
Zhiyi Yu
Dorien Ward‐van Oostwaard
Jacobus PD van Veldhoven
Alessandra Moretti
Karl‐Ludwig Laugwitz
Christine L Mummery
Adriaan P IJzerman
Milena Bellin
author_facet Luca Sala
Zhiyi Yu
Dorien Ward‐van Oostwaard
Jacobus PD van Veldhoven
Alessandra Moretti
Karl‐Ludwig Laugwitz
Christine L Mummery
Adriaan P IJzerman
Milena Bellin
author_sort Luca Sala
collection DOAJ
description Abstract Long‐QT syndrome (LQTS) is an arrhythmogenic disorder characterised by prolongation of the QT interval in the electrocardiogram, which can lead to sudden cardiac death. Pharmacological treatments are far from optimal for congenital forms of LQTS, while the acquired form, often triggered by drugs that (sometimes inadvertently) target the cardiac hERG channel, is still a challenge in drug development because of cardiotoxicity. Current experimental models in vitro fall short in predicting proarrhythmic properties of new drugs in humans. Here, we leveraged a series of isogenically matched, diseased and genetically engineered, human induced pluripotent stem cell‐derived cardiomyocytes (hiPSC‐CMs) from patients to test a novel hERG allosteric modulator for treating congenital LQTS, drug‐induced LQTS or a combination of the two. By slowing IKr deactivation and positively shifting IKr inactivation, the small molecule LUF7346 effectively rescued all of these conditions, demonstrating in a human system that allosteric modulation of hERG may be useful as an approach to treat inherited and drug‐induced LQTS. Furthermore, our study provides experimental support of the value of isogenic pairs of patient hiPSC‐CMs as platforms for testing drug sensitivities and performing safety pharmacology.
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spelling doaj-art-767b30a7cd6e491aa4968dd31eb2d71b2025-08-20T04:03:06ZengSpringer NatureEMBO Molecular Medicine1757-46761757-46842016-07-01891065108110.15252/emmm.201606260A new hERG allosteric modulator rescues genetic and drug‐induced long‐QT syndrome phenotypes in cardiomyocytes from isogenic pairs of patient induced pluripotent stem cellsLuca Sala0Zhiyi Yu1Dorien Ward‐van Oostwaard2Jacobus PD van Veldhoven3Alessandra Moretti4Karl‐Ludwig Laugwitz5Christine L Mummery6Adriaan P IJzerman7Milena Bellin8Department of Anatomy and Embryology, Leiden University Medical CenterGorlaeus Laboratories, Leiden Academic Centre for Drug Research, Leiden UniversityDepartment of Anatomy and Embryology, Leiden University Medical CenterGorlaeus Laboratories, Leiden Academic Centre for Drug Research, Leiden UniversityI. Department of Medicine (Cardiology), Klinikum rechts der Isar, Technical University of MunichI. Department of Medicine (Cardiology), Klinikum rechts der Isar, Technical University of MunichDepartment of Anatomy and Embryology, Leiden University Medical CenterGorlaeus Laboratories, Leiden Academic Centre for Drug Research, Leiden UniversityDepartment of Anatomy and Embryology, Leiden University Medical CenterAbstract Long‐QT syndrome (LQTS) is an arrhythmogenic disorder characterised by prolongation of the QT interval in the electrocardiogram, which can lead to sudden cardiac death. Pharmacological treatments are far from optimal for congenital forms of LQTS, while the acquired form, often triggered by drugs that (sometimes inadvertently) target the cardiac hERG channel, is still a challenge in drug development because of cardiotoxicity. Current experimental models in vitro fall short in predicting proarrhythmic properties of new drugs in humans. Here, we leveraged a series of isogenically matched, diseased and genetically engineered, human induced pluripotent stem cell‐derived cardiomyocytes (hiPSC‐CMs) from patients to test a novel hERG allosteric modulator for treating congenital LQTS, drug‐induced LQTS or a combination of the two. By slowing IKr deactivation and positively shifting IKr inactivation, the small molecule LUF7346 effectively rescued all of these conditions, demonstrating in a human system that allosteric modulation of hERG may be useful as an approach to treat inherited and drug‐induced LQTS. Furthermore, our study provides experimental support of the value of isogenic pairs of patient hiPSC‐CMs as platforms for testing drug sensitivities and performing safety pharmacology.https://doi.org/10.15252/emmm.201606260cardiac arrhythmiadrug screeninghERGhuman induced pluripotent stem cellslong‐QT syndrome
spellingShingle Luca Sala
Zhiyi Yu
Dorien Ward‐van Oostwaard
Jacobus PD van Veldhoven
Alessandra Moretti
Karl‐Ludwig Laugwitz
Christine L Mummery
Adriaan P IJzerman
Milena Bellin
A new hERG allosteric modulator rescues genetic and drug‐induced long‐QT syndrome phenotypes in cardiomyocytes from isogenic pairs of patient induced pluripotent stem cells
EMBO Molecular Medicine
cardiac arrhythmia
drug screening
hERG
human induced pluripotent stem cells
long‐QT syndrome
title A new hERG allosteric modulator rescues genetic and drug‐induced long‐QT syndrome phenotypes in cardiomyocytes from isogenic pairs of patient induced pluripotent stem cells
title_full A new hERG allosteric modulator rescues genetic and drug‐induced long‐QT syndrome phenotypes in cardiomyocytes from isogenic pairs of patient induced pluripotent stem cells
title_fullStr A new hERG allosteric modulator rescues genetic and drug‐induced long‐QT syndrome phenotypes in cardiomyocytes from isogenic pairs of patient induced pluripotent stem cells
title_full_unstemmed A new hERG allosteric modulator rescues genetic and drug‐induced long‐QT syndrome phenotypes in cardiomyocytes from isogenic pairs of patient induced pluripotent stem cells
title_short A new hERG allosteric modulator rescues genetic and drug‐induced long‐QT syndrome phenotypes in cardiomyocytes from isogenic pairs of patient induced pluripotent stem cells
title_sort new herg allosteric modulator rescues genetic and drug induced long qt syndrome phenotypes in cardiomyocytes from isogenic pairs of patient induced pluripotent stem cells
topic cardiac arrhythmia
drug screening
hERG
human induced pluripotent stem cells
long‐QT syndrome
url https://doi.org/10.15252/emmm.201606260
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