The inner nuclear membrane protein, Banf1, has an essential role in triple negative breast cancer cell proliferation and survival

Abstract Triple negative breast cancers (TNBCs) are a heterogenous subcategory of breast cancers which have significantly worse survival outcomes compared to other breast cancer subtypes. However, limited advances have been made towards a targeted TNBC therapeutic and traditional chemotherapies rema...

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Main Authors: Maddison Rose, Joshua T. Burgess, Chee Man Cheong, Iain Richard, Amila Suraweera, Mark N. Adams, Pascal H. G. Duijf, Kenneth J. O’Byrne, Derek J. Richard, Emma Bolderson
Format: Article
Language:English
Published: Nature Portfolio 2025-07-01
Series:Scientific Reports
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Online Access:https://doi.org/10.1038/s41598-025-10346-x
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author Maddison Rose
Joshua T. Burgess
Chee Man Cheong
Iain Richard
Amila Suraweera
Mark N. Adams
Pascal H. G. Duijf
Kenneth J. O’Byrne
Derek J. Richard
Emma Bolderson
author_facet Maddison Rose
Joshua T. Burgess
Chee Man Cheong
Iain Richard
Amila Suraweera
Mark N. Adams
Pascal H. G. Duijf
Kenneth J. O’Byrne
Derek J. Richard
Emma Bolderson
author_sort Maddison Rose
collection DOAJ
description Abstract Triple negative breast cancers (TNBCs) are a heterogenous subcategory of breast cancers which have significantly worse survival outcomes compared to other breast cancer subtypes. However, limited advances have been made towards a targeted TNBC therapeutic and traditional chemotherapies remain the frontline therapy. Therefore, this study aimed to examine the therapeutic potential of a novel TNBC target, Barrier-to-Autointegration Factor 1 (Banf1), including determining Banf1 expression and cellular localisation in non-malignant breast cells and a panel of TNBC cell lines. Bioinformatic analysis of patient samples demonstrated that Banf1 is overexpressed in all breast cancer stages and subtypes. Banf1 depletion inhibited proliferation and induced mitotic arrest in TNBC cells via loss of nuclear envelope integrity and aberrant nuclear morphology, inducing TNBC tumour cell-specific cell death. These findings highlight the significant overexpression and functional involvement of Banf1 in TNBC progression and suggests that it may have potential as a novel anti-cancer target, supporting further investigation.
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institution Kabale University
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publishDate 2025-07-01
publisher Nature Portfolio
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spelling doaj-art-6bbb1b23c0bd4f28b68f59dbc1d3ef1d2025-08-20T03:42:36ZengNature PortfolioScientific Reports2045-23222025-07-0115111310.1038/s41598-025-10346-xThe inner nuclear membrane protein, Banf1, has an essential role in triple negative breast cancer cell proliferation and survivalMaddison Rose0Joshua T. Burgess1Chee Man Cheong2Iain Richard3Amila Suraweera4Mark N. Adams5Pascal H. G. Duijf6Kenneth J. O’Byrne7Derek J. Richard8Emma Bolderson9Cancer Genomics Program, Centre for Genomics and Personalised Health, School of Biomedical Sciences, Faculty of Health, Queensland University of TechnologyCancer Genomics Program, Centre for Genomics and Personalised Health, School of Biomedical Sciences, Faculty of Health, Queensland University of TechnologyCancer Genomics Program, Centre for Genomics and Personalised Health, School of Biomedical Sciences, Faculty of Health, Queensland University of TechnologyCancer Genomics Program, Centre for Genomics and Personalised Health, School of Biomedical Sciences, Faculty of Health, Queensland University of TechnologyCancer Genomics Program, Centre for Genomics and Personalised Health, School of Biomedical Sciences, Faculty of Health, Queensland University of TechnologyCancer Genomics Program, Centre for Genomics and Personalised Health, School of Biomedical Sciences, Faculty of Health, Queensland University of TechnologyCentre for Cancer Biology, University of South Australia & SA PathologyCancer Genomics Program, Centre for Genomics and Personalised Health, School of Biomedical Sciences, Faculty of Health, Queensland University of TechnologyCancer Genomics Program, Centre for Genomics and Personalised Health, School of Biomedical Sciences, Faculty of Health, Queensland University of TechnologyCancer Genomics Program, Centre for Genomics and Personalised Health, School of Biomedical Sciences, Faculty of Health, Queensland University of TechnologyAbstract Triple negative breast cancers (TNBCs) are a heterogenous subcategory of breast cancers which have significantly worse survival outcomes compared to other breast cancer subtypes. However, limited advances have been made towards a targeted TNBC therapeutic and traditional chemotherapies remain the frontline therapy. Therefore, this study aimed to examine the therapeutic potential of a novel TNBC target, Barrier-to-Autointegration Factor 1 (Banf1), including determining Banf1 expression and cellular localisation in non-malignant breast cells and a panel of TNBC cell lines. Bioinformatic analysis of patient samples demonstrated that Banf1 is overexpressed in all breast cancer stages and subtypes. Banf1 depletion inhibited proliferation and induced mitotic arrest in TNBC cells via loss of nuclear envelope integrity and aberrant nuclear morphology, inducing TNBC tumour cell-specific cell death. These findings highlight the significant overexpression and functional involvement of Banf1 in TNBC progression and suggests that it may have potential as a novel anti-cancer target, supporting further investigation.https://doi.org/10.1038/s41598-025-10346-xBreast cancerNuclear envelopeBanf1Mitosis
spellingShingle Maddison Rose
Joshua T. Burgess
Chee Man Cheong
Iain Richard
Amila Suraweera
Mark N. Adams
Pascal H. G. Duijf
Kenneth J. O’Byrne
Derek J. Richard
Emma Bolderson
The inner nuclear membrane protein, Banf1, has an essential role in triple negative breast cancer cell proliferation and survival
Scientific Reports
Breast cancer
Nuclear envelope
Banf1
Mitosis
title The inner nuclear membrane protein, Banf1, has an essential role in triple negative breast cancer cell proliferation and survival
title_full The inner nuclear membrane protein, Banf1, has an essential role in triple negative breast cancer cell proliferation and survival
title_fullStr The inner nuclear membrane protein, Banf1, has an essential role in triple negative breast cancer cell proliferation and survival
title_full_unstemmed The inner nuclear membrane protein, Banf1, has an essential role in triple negative breast cancer cell proliferation and survival
title_short The inner nuclear membrane protein, Banf1, has an essential role in triple negative breast cancer cell proliferation and survival
title_sort inner nuclear membrane protein banf1 has an essential role in triple negative breast cancer cell proliferation and survival
topic Breast cancer
Nuclear envelope
Banf1
Mitosis
url https://doi.org/10.1038/s41598-025-10346-x
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