Dysregulated Coagulation in Parkinson’s Disease

Parkinson’s disease (PD), a prevalent neurodegenerative disorder characterized by dopaminergic neuron degeneration and α-synuclein accumulation, has been increasingly associated with coagulation dysfunction. This review synthesizes emerging evidence linking dysregulated coagulation to PD pathophysio...

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Main Authors: Xinqing Wang, Wenxin Li, Xinyue Zhao, Ning Hu, Xi Wang, Xilin Xiao, Kai Yang, Taolei Sun
Format: Article
Language:English
Published: MDPI AG 2024-11-01
Series:Cells
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Online Access:https://www.mdpi.com/2073-4409/13/22/1874
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author Xinqing Wang
Wenxin Li
Xinyue Zhao
Ning Hu
Xi Wang
Xilin Xiao
Kai Yang
Taolei Sun
author_facet Xinqing Wang
Wenxin Li
Xinyue Zhao
Ning Hu
Xi Wang
Xilin Xiao
Kai Yang
Taolei Sun
author_sort Xinqing Wang
collection DOAJ
description Parkinson’s disease (PD), a prevalent neurodegenerative disorder characterized by dopaminergic neuron degeneration and α-synuclein accumulation, has been increasingly associated with coagulation dysfunction. This review synthesizes emerging evidence linking dysregulated coagulation to PD pathophysiology. We examine the alterations in coagulation parameters, including elevated fibrinogen levels, impaired fibrinolysis, and platelet dysfunction, which collectively contribute to a hypercoagulable state in PD patients. Epidemiological studies have revealed a higher incidence of thrombotic events, such as deep vein thrombosis (DVT) and stroke, among PD patients, suggesting significant comorbidity between PD and coagulation disorders. This review explores the potential pathophysiological mechanisms underlying this association, focusing on the roles of inflammation and oxidative stress. Additionally, we discuss the limitations of current research and propose future directions. This comprehensive analysis underscores the importance of understanding the coagulation–neurodegeneration axis in PD, which may lead to novel diagnostic and therapeutic strategies for this debilitating condition.
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spelling doaj-art-624c7668c0cb4f52b8f3551b63c2f4e72024-11-26T17:56:53ZengMDPI AGCells2073-44092024-11-011322187410.3390/cells13221874Dysregulated Coagulation in Parkinson’s DiseaseXinqing Wang0Wenxin Li1Xinyue Zhao2Ning Hu3Xi Wang4Xilin Xiao5Kai Yang6Taolei Sun7School of Public Health, Hengyang Medical School, University of South China, Hengyang 421001, ChinaSchool of Chemistry, Chemical Engineering and Life Science, Wuhan University of Technology, 122 Luoshi Road, Wuhan 430070, ChinaSchool of Chemistry, Chemical Engineering and Life Science, Wuhan University of Technology, 122 Luoshi Road, Wuhan 430070, ChinaSchool of Chemistry, Chemical Engineering and Life Science, Wuhan University of Technology, 122 Luoshi Road, Wuhan 430070, ChinaSchool of Chemistry, Chemical Engineering and Life Science, Wuhan University of Technology, 122 Luoshi Road, Wuhan 430070, ChinaSchool of Public Health, Hengyang Medical School, University of South China, Hengyang 421001, ChinaSchool of Chemistry, Chemical Engineering and Life Science, Wuhan University of Technology, 122 Luoshi Road, Wuhan 430070, ChinaSchool of Chemistry, Chemical Engineering and Life Science, Wuhan University of Technology, 122 Luoshi Road, Wuhan 430070, ChinaParkinson’s disease (PD), a prevalent neurodegenerative disorder characterized by dopaminergic neuron degeneration and α-synuclein accumulation, has been increasingly associated with coagulation dysfunction. This review synthesizes emerging evidence linking dysregulated coagulation to PD pathophysiology. We examine the alterations in coagulation parameters, including elevated fibrinogen levels, impaired fibrinolysis, and platelet dysfunction, which collectively contribute to a hypercoagulable state in PD patients. Epidemiological studies have revealed a higher incidence of thrombotic events, such as deep vein thrombosis (DVT) and stroke, among PD patients, suggesting significant comorbidity between PD and coagulation disorders. This review explores the potential pathophysiological mechanisms underlying this association, focusing on the roles of inflammation and oxidative stress. Additionally, we discuss the limitations of current research and propose future directions. This comprehensive analysis underscores the importance of understanding the coagulation–neurodegeneration axis in PD, which may lead to novel diagnostic and therapeutic strategies for this debilitating condition.https://www.mdpi.com/2073-4409/13/22/1874coagulation disorderParkinson’s diseaseplateletfibrinolysisdeep veinthrombosis
spellingShingle Xinqing Wang
Wenxin Li
Xinyue Zhao
Ning Hu
Xi Wang
Xilin Xiao
Kai Yang
Taolei Sun
Dysregulated Coagulation in Parkinson’s Disease
Cells
coagulation disorder
Parkinson’s disease
platelet
fibrinolysis
deep vein
thrombosis
title Dysregulated Coagulation in Parkinson’s Disease
title_full Dysregulated Coagulation in Parkinson’s Disease
title_fullStr Dysregulated Coagulation in Parkinson’s Disease
title_full_unstemmed Dysregulated Coagulation in Parkinson’s Disease
title_short Dysregulated Coagulation in Parkinson’s Disease
title_sort dysregulated coagulation in parkinson s disease
topic coagulation disorder
Parkinson’s disease
platelet
fibrinolysis
deep vein
thrombosis
url https://www.mdpi.com/2073-4409/13/22/1874
work_keys_str_mv AT xinqingwang dysregulatedcoagulationinparkinsonsdisease
AT wenxinli dysregulatedcoagulationinparkinsonsdisease
AT xinyuezhao dysregulatedcoagulationinparkinsonsdisease
AT ninghu dysregulatedcoagulationinparkinsonsdisease
AT xiwang dysregulatedcoagulationinparkinsonsdisease
AT xilinxiao dysregulatedcoagulationinparkinsonsdisease
AT kaiyang dysregulatedcoagulationinparkinsonsdisease
AT taoleisun dysregulatedcoagulationinparkinsonsdisease