Bypassing the guardian: regulated cell death pathways in p53-mutant cancers

Abstract Approximately half of all cancers bear mutations in the tumor suppressor p53. Despite decades of research studying p53 function, treatment of p53-mutant cancers remains challenging owing to the effects of p53 mutations on many complex and interrelated signaling networks that promote tumor m...

Full description

Saved in:
Bibliographic Details
Main Authors: Jonathan Y. Chung, Bruce A. Knutson
Format: Article
Language:English
Published: BMC 2025-06-01
Series:Cellular & Molecular Biology Letters
Subjects:
Online Access:https://doi.org/10.1186/s11658-025-00751-5
Tags: Add Tag
No Tags, Be the first to tag this record!
_version_ 1849334611621969920
author Jonathan Y. Chung
Bruce A. Knutson
author_facet Jonathan Y. Chung
Bruce A. Knutson
author_sort Jonathan Y. Chung
collection DOAJ
description Abstract Approximately half of all cancers bear mutations in the tumor suppressor p53. Despite decades of research studying p53 function, treatment of p53-mutant cancers remains challenging owing to the effects of p53 mutations on many complex and interrelated signaling networks that promote tumor metastasis and chemoresistance. Mutations in p53 promote tumor survival by dysregulating cellular homeostasis and preventing activation of regulated cell death (RCD) pathways, which normally promote organismal health by eliminating dysregulated cells. Activation of RCD is a hallmark of effective cancer therapies, and p53-mutant cancers may be particularly susceptible to activation of certain RCD pathways. In this review, we discuss four RCD pathways that are the targets of emerging cancer therapeutics to treat p53-mutant cancers. These RCD pathways include E2F1-dependent apoptosis, necroptosis, mitochondrial permeability transition-driven necrosis, and ferroptosis. We discuss mechanisms of RCD activation, effects of p53 mutation on RCD activation, and current pharmaceutical strategies for RCD activation in p53-mutant cancers. Graphical Abstract
format Article
id doaj-art-61db20009a7346a98e658e5dea0c99b7
institution Kabale University
issn 1689-1392
language English
publishDate 2025-06-01
publisher BMC
record_format Article
series Cellular & Molecular Biology Letters
spelling doaj-art-61db20009a7346a98e658e5dea0c99b72025-08-20T03:45:31ZengBMCCellular & Molecular Biology Letters1689-13922025-06-0130113410.1186/s11658-025-00751-5Bypassing the guardian: regulated cell death pathways in p53-mutant cancersJonathan Y. Chung0Bruce A. Knutson1Department of Biochemistry and Molecular Biology, State University of New York Upstate Medical UniversityDepartment of Biochemistry and Molecular Biology, State University of New York Upstate Medical UniversityAbstract Approximately half of all cancers bear mutations in the tumor suppressor p53. Despite decades of research studying p53 function, treatment of p53-mutant cancers remains challenging owing to the effects of p53 mutations on many complex and interrelated signaling networks that promote tumor metastasis and chemoresistance. Mutations in p53 promote tumor survival by dysregulating cellular homeostasis and preventing activation of regulated cell death (RCD) pathways, which normally promote organismal health by eliminating dysregulated cells. Activation of RCD is a hallmark of effective cancer therapies, and p53-mutant cancers may be particularly susceptible to activation of certain RCD pathways. In this review, we discuss four RCD pathways that are the targets of emerging cancer therapeutics to treat p53-mutant cancers. These RCD pathways include E2F1-dependent apoptosis, necroptosis, mitochondrial permeability transition-driven necrosis, and ferroptosis. We discuss mechanisms of RCD activation, effects of p53 mutation on RCD activation, and current pharmaceutical strategies for RCD activation in p53-mutant cancers. Graphical Abstracthttps://doi.org/10.1186/s11658-025-00751-5P53CancerMetastasisApoptosisNecroptosisFerroptosis
spellingShingle Jonathan Y. Chung
Bruce A. Knutson
Bypassing the guardian: regulated cell death pathways in p53-mutant cancers
Cellular & Molecular Biology Letters
P53
Cancer
Metastasis
Apoptosis
Necroptosis
Ferroptosis
title Bypassing the guardian: regulated cell death pathways in p53-mutant cancers
title_full Bypassing the guardian: regulated cell death pathways in p53-mutant cancers
title_fullStr Bypassing the guardian: regulated cell death pathways in p53-mutant cancers
title_full_unstemmed Bypassing the guardian: regulated cell death pathways in p53-mutant cancers
title_short Bypassing the guardian: regulated cell death pathways in p53-mutant cancers
title_sort bypassing the guardian regulated cell death pathways in p53 mutant cancers
topic P53
Cancer
Metastasis
Apoptosis
Necroptosis
Ferroptosis
url https://doi.org/10.1186/s11658-025-00751-5
work_keys_str_mv AT jonathanychung bypassingtheguardianregulatedcelldeathpathwaysinp53mutantcancers
AT bruceaknutson bypassingtheguardianregulatedcelldeathpathwaysinp53mutantcancers