Mitochondrial maintenance as a novel target for treating steroid-induced osteonecrosis of femoral head: a narrative review

The pathogenesis of steroid-induced osteonecrosis of the femoral head (SONFH) remains unclear; however, emerging evidence suggests that mitochondrial injury plays a significant role. This review aims to elucidate the involvement of mitochondrial dysfunction in SONFH and explore potential therape...

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Main Authors: Yidan Yang, Yi Jian, Youwen Liu, Maoxiao Ma, Jiayi Guo, Bin Xu, Chen Yue
Format: Article
Language:English
Published: Bioscientifica 2024-11-01
Series:EFORT Open Reviews
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Online Access:https://eor.bioscientifica.com/view/journals/eor/9/11/EOR-24-0023.xml
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author Yidan Yang
Yi Jian
Youwen Liu
Maoxiao Ma
Jiayi Guo
Bin Xu
Chen Yue
author_facet Yidan Yang
Yi Jian
Youwen Liu
Maoxiao Ma
Jiayi Guo
Bin Xu
Chen Yue
author_sort Yidan Yang
collection DOAJ
description The pathogenesis of steroid-induced osteonecrosis of the femoral head (SONFH) remains unclear; however, emerging evidence suggests that mitochondrial injury plays a significant role. This review aims to elucidate the involvement of mitochondrial dysfunction in SONFH and explore potential therapeutic targets. A comprehensive literature search was conducted in PubMed, Web of Science, and Elsevier ScienceDirect, focusing on mitochondrial homeostasis, including mitophagy, mitochondrial biogenesis, mitochondrial dynamics, and oxidative stress in SONFH. Ultimately, we included and analyzed a total of 16 studies. Glucocorticoids initially promote but later inhibit mitochondrial biogenesis in osteoblasts, leading to excessive ROS production and mitochondrial dysfunction. This dysfunction impairs osteoblast survival and bone formation, contributing to SONFH progression. Key proteins such as mitochondrial transcription factor A (TFAM) and peroxisome proliferator-activated receptor γ coactivator 1-α (PGC1α) are potential therapeutic targets for promoting mitochondrial biogenesis and reducing ROS-induced damage. Enhancing mitochondrial function and reducing oxidative stress in osteoblasts may prevent or slow the progression of SONFH. Future research should focus on developing these strategies.
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institution Kabale University
issn 2058-5241
language English
publishDate 2024-11-01
publisher Bioscientifica
record_format Article
series EFORT Open Reviews
spelling doaj-art-5dcd40ef3d4d42d9ab4ee43fadca744f2024-11-11T06:49:58ZengBioscientificaEFORT Open Reviews2058-52412024-11-0191110131022https://doi.org/10.1530/EOR-24-0023Mitochondrial maintenance as a novel target for treating steroid-induced osteonecrosis of femoral head: a narrative reviewYidan Yang0Yi Jian1Youwen Liu2Maoxiao Ma3Jiayi Guo4Bin Xu5Chen Yue6Henan University of Chinese Medicine, Zhengzhou, Henan Province, People’s Republic of ChinaDepartment of Orthopedics, Luoyang Orthopedic Hospital of Henan Province, Orthopedic Hospital of Henan Province, Luoyang, Henan Province, People’s Republic of ChinaHenan University of Chinese Medicine, Zhengzhou, Henan Province, People’s Republic of China; Department of Orthopedics, Luoyang Orthopedic Hospital of Henan Province, Orthopedic Hospital of Henan Province, Luoyang, Henan Province, People’s Republic of China Department of Orthopedics, Luoyang Orthopedic Hospital of Henan Province, Orthopedic Hospital of Henan Province, Luoyang, Henan Province, People’s Republic of ChinaHenan University of Chinese Medicine, Zhengzhou, Henan Province, People’s Republic of China; Department of Orthopedics, Luoyang Orthopedic Hospital of Henan Province, Orthopedic Hospital of Henan Province, Luoyang, Henan Province, People’s Republic of China Department of Orthopedics, Tongde Hospital of Zhejiang Province, Hangzhou, Zhejiang Province, People’s Republic of ChinaDepartment of Orthopedics, Luoyang Orthopedic Hospital of Henan Province, Orthopedic Hospital of Henan Province, Luoyang, Henan Province, People’s Republic of China The pathogenesis of steroid-induced osteonecrosis of the femoral head (SONFH) remains unclear; however, emerging evidence suggests that mitochondrial injury plays a significant role. This review aims to elucidate the involvement of mitochondrial dysfunction in SONFH and explore potential therapeutic targets. A comprehensive literature search was conducted in PubMed, Web of Science, and Elsevier ScienceDirect, focusing on mitochondrial homeostasis, including mitophagy, mitochondrial biogenesis, mitochondrial dynamics, and oxidative stress in SONFH. Ultimately, we included and analyzed a total of 16 studies. Glucocorticoids initially promote but later inhibit mitochondrial biogenesis in osteoblasts, leading to excessive ROS production and mitochondrial dysfunction. This dysfunction impairs osteoblast survival and bone formation, contributing to SONFH progression. Key proteins such as mitochondrial transcription factor A (TFAM) and peroxisome proliferator-activated receptor γ coactivator 1-α (PGC1α) are potential therapeutic targets for promoting mitochondrial biogenesis and reducing ROS-induced damage. Enhancing mitochondrial function and reducing oxidative stress in osteoblasts may prevent or slow the progression of SONFH. Future research should focus on developing these strategies. https://eor.bioscientifica.com/view/journals/eor/9/11/EOR-24-0023.xmlmitochondria dynamicsmitochondrial homeostasismitophagysteroid-induced osteonecrosis of the femoral head
spellingShingle Yidan Yang
Yi Jian
Youwen Liu
Maoxiao Ma
Jiayi Guo
Bin Xu
Chen Yue
Mitochondrial maintenance as a novel target for treating steroid-induced osteonecrosis of femoral head: a narrative review
EFORT Open Reviews
mitochondria dynamics
mitochondrial homeostasis
mitophagy
steroid-induced osteonecrosis of the femoral head
title Mitochondrial maintenance as a novel target for treating steroid-induced osteonecrosis of femoral head: a narrative review
title_full Mitochondrial maintenance as a novel target for treating steroid-induced osteonecrosis of femoral head: a narrative review
title_fullStr Mitochondrial maintenance as a novel target for treating steroid-induced osteonecrosis of femoral head: a narrative review
title_full_unstemmed Mitochondrial maintenance as a novel target for treating steroid-induced osteonecrosis of femoral head: a narrative review
title_short Mitochondrial maintenance as a novel target for treating steroid-induced osteonecrosis of femoral head: a narrative review
title_sort mitochondrial maintenance as a novel target for treating steroid induced osteonecrosis of femoral head a narrative review
topic mitochondria dynamics
mitochondrial homeostasis
mitophagy
steroid-induced osteonecrosis of the femoral head
url https://eor.bioscientifica.com/view/journals/eor/9/11/EOR-24-0023.xml
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