Searching for the ‘X’ factor: investigating the genetics of primary ovarian insufficiency

Abstract Primary ovarian insufficiency (POI) is the cessation of ovarian function before the age of 40. The causes of POI are heterogeneous, but substantial evidence exists to support a genetic basis of POI, particularly in the critical involvement of genes on the X chromosome. Recent studies have r...

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Main Authors: Anya Knight, Sara Sugin, Andrea Jurisicova
Format: Article
Language:English
Published: BMC 2024-11-01
Series:Journal of Ovarian Research
Subjects:
Online Access:https://doi.org/10.1186/s13048-024-01555-5
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author Anya Knight
Sara Sugin
Andrea Jurisicova
author_facet Anya Knight
Sara Sugin
Andrea Jurisicova
author_sort Anya Knight
collection DOAJ
description Abstract Primary ovarian insufficiency (POI) is the cessation of ovarian function before the age of 40. The causes of POI are heterogeneous, but substantial evidence exists to support a genetic basis of POI, particularly in the critical involvement of genes on the X chromosome. Recent studies have revealed novel candidate genes through the identification of copy number variations associated with POI. This review summarizes the genes located on the X chromosome with variants shown to be associated with POI in humans and/or in mice. Additionally, we present evidence to support the potential involvement of these candidate genes in the etiology of POI. We conducted a literature search in PubMed to identify case studies and screenings for the genetic causes of POI. We then performed systematic searches for the proposed candidate genes to investigate their potential reproductive roles. Of the X-linked candidate genes investigated, 10 were found to have variants associated with cases of POI in humans. An additional 10 genes were found to play a supportive role in POI. Other genes were not implicated in any cases of POI but were associated with various roles in reproduction. In the majority of cases where variants were identified through whole-exome sequencing, rather than targeted screening of candidate genes, more than one genetic variant was identified. Overall, this review supports past findings that the X chromosome plays a critical role in ovarian function, as demonstrated by a link between POI and various disruptions to genes on the X chromosome. Current genetic screening for POI, which includes only FMR1, is inadequate to capture the majority of cases with a genetic origin. An expanded genetic testing may improve health outcomes for individuals with POI as it could lead to better early interventions and education about these health risks.
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spelling doaj-art-4a91bd0e80934b0e9b0e2d51953668de2024-12-01T12:40:07ZengBMCJournal of Ovarian Research1757-22152024-11-0117111610.1186/s13048-024-01555-5Searching for the ‘X’ factor: investigating the genetics of primary ovarian insufficiencyAnya Knight0Sara Sugin1Andrea Jurisicova2Department of Physiology, Temerty Faculty of Medicine, University of TorontoDepartment of Physiology, Temerty Faculty of Medicine, University of TorontoDepartment of Physiology, Temerty Faculty of Medicine, University of TorontoAbstract Primary ovarian insufficiency (POI) is the cessation of ovarian function before the age of 40. The causes of POI are heterogeneous, but substantial evidence exists to support a genetic basis of POI, particularly in the critical involvement of genes on the X chromosome. Recent studies have revealed novel candidate genes through the identification of copy number variations associated with POI. This review summarizes the genes located on the X chromosome with variants shown to be associated with POI in humans and/or in mice. Additionally, we present evidence to support the potential involvement of these candidate genes in the etiology of POI. We conducted a literature search in PubMed to identify case studies and screenings for the genetic causes of POI. We then performed systematic searches for the proposed candidate genes to investigate their potential reproductive roles. Of the X-linked candidate genes investigated, 10 were found to have variants associated with cases of POI in humans. An additional 10 genes were found to play a supportive role in POI. Other genes were not implicated in any cases of POI but were associated with various roles in reproduction. In the majority of cases where variants were identified through whole-exome sequencing, rather than targeted screening of candidate genes, more than one genetic variant was identified. Overall, this review supports past findings that the X chromosome plays a critical role in ovarian function, as demonstrated by a link between POI and various disruptions to genes on the X chromosome. Current genetic screening for POI, which includes only FMR1, is inadequate to capture the majority of cases with a genetic origin. An expanded genetic testing may improve health outcomes for individuals with POI as it could lead to better early interventions and education about these health risks.https://doi.org/10.1186/s13048-024-01555-5Primary ovarian insufficiencyX chromosome inactivationGene dosageFolliculogenesisOogenesisOvarian phenotype
spellingShingle Anya Knight
Sara Sugin
Andrea Jurisicova
Searching for the ‘X’ factor: investigating the genetics of primary ovarian insufficiency
Journal of Ovarian Research
Primary ovarian insufficiency
X chromosome inactivation
Gene dosage
Folliculogenesis
Oogenesis
Ovarian phenotype
title Searching for the ‘X’ factor: investigating the genetics of primary ovarian insufficiency
title_full Searching for the ‘X’ factor: investigating the genetics of primary ovarian insufficiency
title_fullStr Searching for the ‘X’ factor: investigating the genetics of primary ovarian insufficiency
title_full_unstemmed Searching for the ‘X’ factor: investigating the genetics of primary ovarian insufficiency
title_short Searching for the ‘X’ factor: investigating the genetics of primary ovarian insufficiency
title_sort searching for the x factor investigating the genetics of primary ovarian insufficiency
topic Primary ovarian insufficiency
X chromosome inactivation
Gene dosage
Folliculogenesis
Oogenesis
Ovarian phenotype
url https://doi.org/10.1186/s13048-024-01555-5
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