Galectin-1 overexpression induces normal fibroblasts translate into cancer-associated fibroblasts and attenuates the sensitivity of anlotinib in lung cancer

We aimed to investigate galectin-1 overexpression induces normal fibroblasts (NFs) translates into cancer-associated fibroblasts (CAFs). Galectin-1 overexpression was conducted in Human embryonic lung fibroblasts (HFL1) cell. The motilities of H1299 and A549 cells were measured. Human umbilical vein...

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Main Authors: Lei Zhang, Wenbang Chen, Xiaojun Li, Gengming Wang, Fubao Xing, Xiao Zhu
Format: Article
Language:English
Published: Taylor & Francis Group 2024-12-01
Series:Cell Adhesion & Migration
Subjects:
Online Access:https://www.tandfonline.com/doi/10.1080/19336918.2024.2335881
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author Lei Zhang
Wenbang Chen
Xiaojun Li
Gengming Wang
Fubao Xing
Xiao Zhu
author_facet Lei Zhang
Wenbang Chen
Xiaojun Li
Gengming Wang
Fubao Xing
Xiao Zhu
author_sort Lei Zhang
collection DOAJ
description We aimed to investigate galectin-1 overexpression induces normal fibroblasts (NFs) translates into cancer-associated fibroblasts (CAFs). Galectin-1 overexpression was conducted in Human embryonic lung fibroblasts (HFL1) cell. The motilities of H1299 and A549 cells were measured. Human umbilical vein endothelial cell (HUVEC) proliferation and tube formation ability were assessed. Tumor volume and tumor weight was recorded. Cells motilities were increased, while apoptosis rates were decreased after CMs co-cultured. B-cell lymphoma-2 (Bcl-2) expression level was increased, while Bcl2-associatedX (Bax) and cleaved-caspase3 decreased. CMs treatment enhanced HUVEC proliferation and tube formation. Tumor volume and weight in CMs treated mice were increased, and the sensitivity of anlotinib in co-cultured cells was decreased. Our results revealed that galectin-1 overexpression induced NFs translated into CAFs.
format Article
id doaj-art-45bd411b7a7e47d5995a37643de20f3f
institution Kabale University
issn 1933-6918
1933-6926
language English
publishDate 2024-12-01
publisher Taylor & Francis Group
record_format Article
series Cell Adhesion & Migration
spelling doaj-art-45bd411b7a7e47d5995a37643de20f3f2024-11-25T08:41:13ZengTaylor & Francis GroupCell Adhesion & Migration1933-69181933-69262024-12-0118111110.1080/19336918.2024.2335881Galectin-1 overexpression induces normal fibroblasts translate into cancer-associated fibroblasts and attenuates the sensitivity of anlotinib in lung cancerLei Zhang0Wenbang Chen1Xiaojun Li2Gengming Wang3Fubao Xing4Xiao Zhu50Department of Thoracic Surgery, The First Affiliated Hospital of Bengbu Medical College, Bengbu, Anhui, China0Department of Thoracic Surgery, The First Affiliated Hospital of Bengbu Medical College, Bengbu, Anhui, China0Department of Thoracic Surgery, The First Affiliated Hospital of Bengbu Medical College, Bengbu, Anhui, ChinaDepartment of Radiotherapy, The First Affiliated Hospital of Bengbu Medical College, Bengbu, Anhui, China0Department of Thoracic Surgery, The First Affiliated Hospital of Bengbu Medical College, Bengbu, Anhui, China0Department of Thoracic Surgery, The First Affiliated Hospital of Bengbu Medical College, Bengbu, Anhui, ChinaWe aimed to investigate galectin-1 overexpression induces normal fibroblasts (NFs) translates into cancer-associated fibroblasts (CAFs). Galectin-1 overexpression was conducted in Human embryonic lung fibroblasts (HFL1) cell. The motilities of H1299 and A549 cells were measured. Human umbilical vein endothelial cell (HUVEC) proliferation and tube formation ability were assessed. Tumor volume and tumor weight was recorded. Cells motilities were increased, while apoptosis rates were decreased after CMs co-cultured. B-cell lymphoma-2 (Bcl-2) expression level was increased, while Bcl2-associatedX (Bax) and cleaved-caspase3 decreased. CMs treatment enhanced HUVEC proliferation and tube formation. Tumor volume and weight in CMs treated mice were increased, and the sensitivity of anlotinib in co-cultured cells was decreased. Our results revealed that galectin-1 overexpression induced NFs translated into CAFs.https://www.tandfonline.com/doi/10.1080/19336918.2024.2335881Condition mediumsHUVECTGF-β1tube formationα-SMA
spellingShingle Lei Zhang
Wenbang Chen
Xiaojun Li
Gengming Wang
Fubao Xing
Xiao Zhu
Galectin-1 overexpression induces normal fibroblasts translate into cancer-associated fibroblasts and attenuates the sensitivity of anlotinib in lung cancer
Cell Adhesion & Migration
Condition mediums
HUVEC
TGF-β1
tube formation
α-SMA
title Galectin-1 overexpression induces normal fibroblasts translate into cancer-associated fibroblasts and attenuates the sensitivity of anlotinib in lung cancer
title_full Galectin-1 overexpression induces normal fibroblasts translate into cancer-associated fibroblasts and attenuates the sensitivity of anlotinib in lung cancer
title_fullStr Galectin-1 overexpression induces normal fibroblasts translate into cancer-associated fibroblasts and attenuates the sensitivity of anlotinib in lung cancer
title_full_unstemmed Galectin-1 overexpression induces normal fibroblasts translate into cancer-associated fibroblasts and attenuates the sensitivity of anlotinib in lung cancer
title_short Galectin-1 overexpression induces normal fibroblasts translate into cancer-associated fibroblasts and attenuates the sensitivity of anlotinib in lung cancer
title_sort galectin 1 overexpression induces normal fibroblasts translate into cancer associated fibroblasts and attenuates the sensitivity of anlotinib in lung cancer
topic Condition mediums
HUVEC
TGF-β1
tube formation
α-SMA
url https://www.tandfonline.com/doi/10.1080/19336918.2024.2335881
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AT wenbangchen galectin1overexpressioninducesnormalfibroblaststranslateintocancerassociatedfibroblastsandattenuatesthesensitivityofanlotinibinlungcancer
AT xiaojunli galectin1overexpressioninducesnormalfibroblaststranslateintocancerassociatedfibroblastsandattenuatesthesensitivityofanlotinibinlungcancer
AT gengmingwang galectin1overexpressioninducesnormalfibroblaststranslateintocancerassociatedfibroblastsandattenuatesthesensitivityofanlotinibinlungcancer
AT fubaoxing galectin1overexpressioninducesnormalfibroblaststranslateintocancerassociatedfibroblastsandattenuatesthesensitivityofanlotinibinlungcancer
AT xiaozhu galectin1overexpressioninducesnormalfibroblaststranslateintocancerassociatedfibroblastsandattenuatesthesensitivityofanlotinibinlungcancer