α-Bisabolol alleviates diesel exhaust particle-induced lung injury and mitochondrial dysfunction by regulating inflammatory, oxidative stress, and apoptotic biomarkers through the c-Jun N-terminal kinase signaling pathway

α-Bisabolol alleviates diesel exhaust particle-induced lung injury and mitochondrial dysfunction by regulating inflammatory, oxidative stress, and apoptotic biomarkers through the c-Jun N-terminal kinase signaling pathway

IntroductionExposure to particulate matter ≤2.5 μm in diameter (PM2.5) is associated with adverse respiratory outcomes, including alterations to lung morphology and function. These associations were reported even at concentrations lower than the current annual limit of PM2.5. Inhalation of PM2.5, of...

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Bibliographic Details
Main Authors: Sumaya Beegam, Nur Elena Zaaba, Ozaz Elzaki, Abderrahim Nemmar
Format: Article
Language:English
Published: Frontiers Media S.A. 2025-01-01
Series:Frontiers in Pharmacology
Subjects:
diesel exhaust particles
α-bisabolol
airway resistance
oxidative stress
inflammation
mitochondrial dysfunction
Online Access:https://www.frontiersin.org/articles/10.3389/fphar.2024.1485101/full
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