Population PK modeling of certolizumab pegol in pregnant women with chronic inflammatory diseases

Abstract Certolizumab pegol (CZP; CIMZIA™) is the only Fc‐free tumor necrosis factor inhibitor with data from a clinical study demonstrating no to minimal placental transfer. The pharmacokinetics (PK) of certolizumab pegol during pregnancy and postpartum in women with chronic inflammatory diseases w...

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Main Authors: Denis Menshykau, Jagdev Sidhu, Laura Shaughnessy, Rocio Lledo‐Garcia, Pinky Dua, Marie Teil, Akash Khandelwal
Format: Article
Language:English
Published: Wiley 2024-11-01
Series:CPT: Pharmacometrics & Systems Pharmacology
Online Access:https://doi.org/10.1002/psp4.13220
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author Denis Menshykau
Jagdev Sidhu
Laura Shaughnessy
Rocio Lledo‐Garcia
Pinky Dua
Marie Teil
Akash Khandelwal
author_facet Denis Menshykau
Jagdev Sidhu
Laura Shaughnessy
Rocio Lledo‐Garcia
Pinky Dua
Marie Teil
Akash Khandelwal
author_sort Denis Menshykau
collection DOAJ
description Abstract Certolizumab pegol (CZP; CIMZIA™) is the only Fc‐free tumor necrosis factor inhibitor with data from a clinical study demonstrating no to minimal placental transfer. The pharmacokinetics (PK) of certolizumab pegol during pregnancy and postpartum in women with chronic inflammatory diseases were assessed using a population PK model based on data from the CHERISH study (NCT04163016), a longitudinal, prospective, open‐label PK phase IB study. Model development was performed in NONMEM using a frequentist prior approach, with prior information based on a population PK model for certolizumab pegol in non‐pregnant adult patients (NCT04740814). A one‐compartment model with first‐order absorption (Ka = 0.236 1/day) and linear elimination (CL/F = 0.416 L/day) from the central compartment (V/F = 7.86 L) best described certolizumab pegol PK in the CHERISH study. The structural model parameters were estimated with good precision (RSE < 25%). Baseline BW was included as a covariate on CL/F and V/F. Pregnancy trimester and time‐varying log‐transformed anti‐drug antibody (ADA) titer were identified as the only significant covariates for CL/F with a comparable influence on CL/F. Individuals with higher ADA titer (75th percentile) during pregnancy exhibited CL/F up to 1.43‐fold higher relative to individuals postpartum that showed median levels of ADA titer. However, the confidence interval for the combined effect of pregnancy stage and ADA titer effects on CL/F overlapped with the CL/F range of the typical individual postpartum. In addition, simulations showed a large overlap in certolizumab pegol concentrations between pregnant and non‐pregnant adults. The findings of this population PK analysis support the maintenance of established certolizumab pegol dosing regimens throughout pregnancy.
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spelling doaj-art-0ea6f96c831248acb8c94d0a71f46dd02024-11-20T17:18:44ZengWileyCPT: Pharmacometrics & Systems Pharmacology2163-83062024-11-0113111904191410.1002/psp4.13220Population PK modeling of certolizumab pegol in pregnant women with chronic inflammatory diseasesDenis Menshykau0Jagdev Sidhu1Laura Shaughnessy2Rocio Lledo‐Garcia3Pinky Dua4Marie Teil5Akash Khandelwal6UCB Biosciences GmbH Monheim am Rhein GermanyUCB Australia Pty Ltd Malvern Victoria AustraliaUCB BioSciences Inc Raleigh North Carolina USAUCB Pharma Slough UKUCB Pharma Slough UKUCB Biopharma SRL Brussels BelgiumUCB Biosciences GmbH Monheim am Rhein GermanyAbstract Certolizumab pegol (CZP; CIMZIA™) is the only Fc‐free tumor necrosis factor inhibitor with data from a clinical study demonstrating no to minimal placental transfer. The pharmacokinetics (PK) of certolizumab pegol during pregnancy and postpartum in women with chronic inflammatory diseases were assessed using a population PK model based on data from the CHERISH study (NCT04163016), a longitudinal, prospective, open‐label PK phase IB study. Model development was performed in NONMEM using a frequentist prior approach, with prior information based on a population PK model for certolizumab pegol in non‐pregnant adult patients (NCT04740814). A one‐compartment model with first‐order absorption (Ka = 0.236 1/day) and linear elimination (CL/F = 0.416 L/day) from the central compartment (V/F = 7.86 L) best described certolizumab pegol PK in the CHERISH study. The structural model parameters were estimated with good precision (RSE < 25%). Baseline BW was included as a covariate on CL/F and V/F. Pregnancy trimester and time‐varying log‐transformed anti‐drug antibody (ADA) titer were identified as the only significant covariates for CL/F with a comparable influence on CL/F. Individuals with higher ADA titer (75th percentile) during pregnancy exhibited CL/F up to 1.43‐fold higher relative to individuals postpartum that showed median levels of ADA titer. However, the confidence interval for the combined effect of pregnancy stage and ADA titer effects on CL/F overlapped with the CL/F range of the typical individual postpartum. In addition, simulations showed a large overlap in certolizumab pegol concentrations between pregnant and non‐pregnant adults. The findings of this population PK analysis support the maintenance of established certolizumab pegol dosing regimens throughout pregnancy.https://doi.org/10.1002/psp4.13220
spellingShingle Denis Menshykau
Jagdev Sidhu
Laura Shaughnessy
Rocio Lledo‐Garcia
Pinky Dua
Marie Teil
Akash Khandelwal
Population PK modeling of certolizumab pegol in pregnant women with chronic inflammatory diseases
CPT: Pharmacometrics & Systems Pharmacology
title Population PK modeling of certolizumab pegol in pregnant women with chronic inflammatory diseases
title_full Population PK modeling of certolizumab pegol in pregnant women with chronic inflammatory diseases
title_fullStr Population PK modeling of certolizumab pegol in pregnant women with chronic inflammatory diseases
title_full_unstemmed Population PK modeling of certolizumab pegol in pregnant women with chronic inflammatory diseases
title_short Population PK modeling of certolizumab pegol in pregnant women with chronic inflammatory diseases
title_sort population pk modeling of certolizumab pegol in pregnant women with chronic inflammatory diseases
url https://doi.org/10.1002/psp4.13220
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