Inhibiting CD36 palmitoylation improves cardiac function post-infarction by regulating lipid metabolic homeostasis and autophagy

Inhibiting CD36 palmitoylation improves cardiac function post-infarction by regulating lipid metabolic homeostasis and autophagy

Abstract Alterations in myocardial energy substrate metabolism and mitochondrial injury following myocardial infarction (MI) lead to structural and functional abnormalities of the heart. The fatty acid translocase CD36 (CD36) plays a pivotal role in regulating lipid homeostasis and mitochondrial met...

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Main Authors: Qingwei Zhang, Jiamin Li, Xin Liu, Ximing Chen, Liwei Zhu, Zhen Zhang, Yingying Hu, Tong Zhao, Han Lou, Henghui Xu, Wenjie Zhao, Xinxin Dong, Zeqi Sun, Xiuxiu Sun, Baofeng Yang, Yong Zhang
Format: Article
Language:English
Published: Nature Portfolio 2025-07-01
Series:Nature Communications
Online Access:https://doi.org/10.1038/s41467-025-61875-y
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https://doi.org/10.1038/s41467-025-61875-y

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