RNase 7 and Th cytokines synergistically increase the secretion of interleukin-6 from keratinocytes

Abstract Epidermal keratinocytes are a major source of antimicrobial peptides and proteins. RNase 7 (R7) exhibits antimicrobial and immunomodulatory activities and is upregulated in the lesional skin of atopic dermatitis and psoriasis patients. Here, we performed mRNA microarray analysis to study th...

Full description

Saved in:
Bibliographic Details
Main Authors: Shruti Chopra, Janine Siegl, Verena Kopfnagel, Sylvia Dreyer, Franziska Rademacher, Juliane Lokau, Christoph Garbers, Jürgen Harder, Thomas Werfel, Katinka Döhner
Format: Article
Language:English
Published: Nature Portfolio 2025-06-01
Series:Scientific Reports
Subjects:
Online Access:https://doi.org/10.1038/s41598-025-04403-8
Tags: Add Tag
No Tags, Be the first to tag this record!
_version_ 1849724886518333440
author Shruti Chopra
Janine Siegl
Verena Kopfnagel
Sylvia Dreyer
Franziska Rademacher
Juliane Lokau
Christoph Garbers
Jürgen Harder
Thomas Werfel
Katinka Döhner
author_facet Shruti Chopra
Janine Siegl
Verena Kopfnagel
Sylvia Dreyer
Franziska Rademacher
Juliane Lokau
Christoph Garbers
Jürgen Harder
Thomas Werfel
Katinka Döhner
author_sort Shruti Chopra
collection DOAJ
description Abstract Epidermal keratinocytes are a major source of antimicrobial peptides and proteins. RNase 7 (R7) exhibits antimicrobial and immunomodulatory activities and is upregulated in the lesional skin of atopic dermatitis and psoriasis patients. Here, we performed mRNA microarray analysis to study the influence of R7 on gene expression in human keratinocytes. R7 induced several genes encoding proteins potentially involved in skin integrity and inflammation, including IL-24. Since IL-24 is involved in the pathogenesis of inflammatory skin diseases and since it is induced by IL-6, we further characterized the effect of R7 on IL-6 and IL-24 expression and secretion. R7 increased the release of the cytokines IL-6 and IL-24 by keratinocytes. Using a function-blocking antibody against IL-6 we showed that the R7-induced IL-24 secretion was triggered by IL-6. IL-6 released from R7-stimulated keratinocytes induced STAT3 signaling in target keratinocytes, suggesting a potential pathway resulting in IL-24 induction. Pre-stimulation with IL-17 or IFN-γ, cytokines involved in the pathogenesis of psoriasis, further enhanced R7-induced IL-6 secretion. R7 increased IL-6 secretion mainly from undifferentiated keratinocytes, suggesting that in the epidermis, R7 released from the stratum granulosum may induce expression and secretion of cytokines from basal undifferentiated keratinocytes. In conclusion, our data show that R7 enhances the release of IL-6 in undifferentiated primary keratinocytes, which may have consequences for skin inflammation, defense against pathogens, and wound healing.
format Article
id doaj-art-0d0edda5744c48c29aae747fc9bafc80
institution DOAJ
issn 2045-2322
language English
publishDate 2025-06-01
publisher Nature Portfolio
record_format Article
series Scientific Reports
spelling doaj-art-0d0edda5744c48c29aae747fc9bafc802025-08-20T03:10:36ZengNature PortfolioScientific Reports2045-23222025-06-0115111510.1038/s41598-025-04403-8RNase 7 and Th cytokines synergistically increase the secretion of interleukin-6 from keratinocytesShruti Chopra0Janine Siegl1Verena Kopfnagel2Sylvia Dreyer3Franziska Rademacher4Juliane Lokau5Christoph Garbers6Jürgen Harder7Thomas Werfel8Katinka Döhner9Department of Dermatology and Allergy, Hannover Medical SchoolDepartment of Dermatology and Allergy, Hannover Medical SchoolDepartment of Dermatology and Allergy, Hannover Medical SchoolDepartment of Dermatology and Allergy, Hannover Medical SchoolDepartment of Dermatology, Quincke Research Center, Kiel UniversityInstitute of Clinical Biochemistry, Hannover Medical SchoolInstitute of Clinical Biochemistry, Hannover Medical SchoolDepartment of Dermatology, Quincke Research Center, Kiel UniversityDepartment of Dermatology and Allergy, Hannover Medical SchoolDepartment of Dermatology and Allergy, Hannover Medical SchoolAbstract Epidermal keratinocytes are a major source of antimicrobial peptides and proteins. RNase 7 (R7) exhibits antimicrobial and immunomodulatory activities and is upregulated in the lesional skin of atopic dermatitis and psoriasis patients. Here, we performed mRNA microarray analysis to study the influence of R7 on gene expression in human keratinocytes. R7 induced several genes encoding proteins potentially involved in skin integrity and inflammation, including IL-24. Since IL-24 is involved in the pathogenesis of inflammatory skin diseases and since it is induced by IL-6, we further characterized the effect of R7 on IL-6 and IL-24 expression and secretion. R7 increased the release of the cytokines IL-6 and IL-24 by keratinocytes. Using a function-blocking antibody against IL-6 we showed that the R7-induced IL-24 secretion was triggered by IL-6. IL-6 released from R7-stimulated keratinocytes induced STAT3 signaling in target keratinocytes, suggesting a potential pathway resulting in IL-24 induction. Pre-stimulation with IL-17 or IFN-γ, cytokines involved in the pathogenesis of psoriasis, further enhanced R7-induced IL-6 secretion. R7 increased IL-6 secretion mainly from undifferentiated keratinocytes, suggesting that in the epidermis, R7 released from the stratum granulosum may induce expression and secretion of cytokines from basal undifferentiated keratinocytes. In conclusion, our data show that R7 enhances the release of IL-6 in undifferentiated primary keratinocytes, which may have consequences for skin inflammation, defense against pathogens, and wound healing.https://doi.org/10.1038/s41598-025-04403-8RNase 7IL-6IL-24IL-17IFN-γInflammatory skin diseases
spellingShingle Shruti Chopra
Janine Siegl
Verena Kopfnagel
Sylvia Dreyer
Franziska Rademacher
Juliane Lokau
Christoph Garbers
Jürgen Harder
Thomas Werfel
Katinka Döhner
RNase 7 and Th cytokines synergistically increase the secretion of interleukin-6 from keratinocytes
Scientific Reports
RNase 7
IL-6
IL-24
IL-17
IFN-γ
Inflammatory skin diseases
title RNase 7 and Th cytokines synergistically increase the secretion of interleukin-6 from keratinocytes
title_full RNase 7 and Th cytokines synergistically increase the secretion of interleukin-6 from keratinocytes
title_fullStr RNase 7 and Th cytokines synergistically increase the secretion of interleukin-6 from keratinocytes
title_full_unstemmed RNase 7 and Th cytokines synergistically increase the secretion of interleukin-6 from keratinocytes
title_short RNase 7 and Th cytokines synergistically increase the secretion of interleukin-6 from keratinocytes
title_sort rnase 7 and th cytokines synergistically increase the secretion of interleukin 6 from keratinocytes
topic RNase 7
IL-6
IL-24
IL-17
IFN-γ
Inflammatory skin diseases
url https://doi.org/10.1038/s41598-025-04403-8
work_keys_str_mv AT shrutichopra rnase7andthcytokinessynergisticallyincreasethesecretionofinterleukin6fromkeratinocytes
AT janinesiegl rnase7andthcytokinessynergisticallyincreasethesecretionofinterleukin6fromkeratinocytes
AT verenakopfnagel rnase7andthcytokinessynergisticallyincreasethesecretionofinterleukin6fromkeratinocytes
AT sylviadreyer rnase7andthcytokinessynergisticallyincreasethesecretionofinterleukin6fromkeratinocytes
AT franziskarademacher rnase7andthcytokinessynergisticallyincreasethesecretionofinterleukin6fromkeratinocytes
AT julianelokau rnase7andthcytokinessynergisticallyincreasethesecretionofinterleukin6fromkeratinocytes
AT christophgarbers rnase7andthcytokinessynergisticallyincreasethesecretionofinterleukin6fromkeratinocytes
AT jurgenharder rnase7andthcytokinessynergisticallyincreasethesecretionofinterleukin6fromkeratinocytes
AT thomaswerfel rnase7andthcytokinessynergisticallyincreasethesecretionofinterleukin6fromkeratinocytes
AT katinkadohner rnase7andthcytokinessynergisticallyincreasethesecretionofinterleukin6fromkeratinocytes