Alkali burn injury model of meibomian gland dysfunction in mice

AIM: To establish a stable, short-time, low-cost and reliable murine model of meibomian gland dysfunction (MGD). METHODS: A filter paper sheet soaked in 1.0 mol/L sodium hydroxide (NaOH) solution was used to touch the eyelid margin of C57BL/6J mice for 10s to establish the model. The other eye was l...

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Main Authors: Yong Li, Ya-Qiong Yang, Yong Lin, Ke Yan, Yu-Fei Lyu, Zhao-Qiang Zhang, Cai-Hong Huang, Jiao-Yue Hu, Zu-Guo Liu
Format: Article
Language:English
Published: Press of International Journal of Ophthalmology (IJO PRESS) 2024-12-01
Series:International Journal of Ophthalmology
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Online Access:http://ies.ijo.cn/en_publish/2024/12/20241202.pdf
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author Yong Li
Ya-Qiong Yang
Yong Lin
Ke Yan
Yu-Fei Lyu
Zhao-Qiang Zhang
Cai-Hong Huang
Jiao-Yue Hu
Zu-Guo Liu
author_facet Yong Li
Ya-Qiong Yang
Yong Lin
Ke Yan
Yu-Fei Lyu
Zhao-Qiang Zhang
Cai-Hong Huang
Jiao-Yue Hu
Zu-Guo Liu
author_sort Yong Li
collection DOAJ
description AIM: To establish a stable, short-time, low-cost and reliable murine model of meibomian gland dysfunction (MGD). METHODS: A filter paper sheet soaked in 1.0 mol/L sodium hydroxide (NaOH) solution was used to touch the eyelid margin of C57BL/6J mice for 10s to establish the model. The other eye was left untreated as a control group. Eyelid margin morphological changes and the meibomian glands (MGs) were observed by slit lamp microscopy on days 5 and 10 post-burn. Hematoxylin-eosin (HE) staining and Oil red O staining were adopted in detecting the changes in MGs morphology and lipid deposition. Real-time polymerase chain reaction, Western blot, immunofluorescence staining and immunohistochemical staining were used to detect interleukin (IL)-6, IL-1β, IL-18, tumor necroses factor (TNF)-α, interferon (IFN)-γ, nicotinamide adenine dinucleotide phosphate (NADPH) oxidase 4 (NOX4), 3-nitroturosine (3-NT), 4-hydroxynonenal (4-HNE) and cytokeratin 10 (K10) expression changes in MGs. RESULTS: MGs showed plugging of orifice, glandular deficiency, abnormal acinar morphology, ductal dilatation, and lipid deposition after alkali burn. The expressions of IL-6, IL-18, IL-1β, IFN-γ, and TNF-α indicators of inflammation and oxidative stress in MGs tissues were significantly increased. Abnormal keratinization increased in the MG duct. CONCLUSION: A murine model of MGD is established by alkali burn of the eyelid margin that matches the clinical presentation of MGD providing a stable, short-time, low-cost, and reliable MGD model. The new method suggests efficient avenues for future research.
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publishDate 2024-12-01
publisher Press of International Journal of Ophthalmology (IJO PRESS)
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spelling doaj-art-05cb4e9c6b4641e782b02d94d9ca79d92024-11-19T03:13:35ZengPress of International Journal of Ophthalmology (IJO PRESS)International Journal of Ophthalmology2222-39592227-48982024-12-0117122158216610.18240/ijo.2024.12.0220241202Alkali burn injury model of meibomian gland dysfunction in miceYong Li0Ya-Qiong Yang1Yong Lin2Ke Yan3Yu-Fei Lyu4Zhao-Qiang Zhang5Cai-Hong Huang6Jiao-Yue Hu7Zu-Guo Liu8Zu-Guo Liu. Key Laboratory of Ophthalmology, University of South China, Hengyang 421000, Hunan Province, China. zuguoliu@xmu.edu.cnXiamen University Affiliated Xiamen Eye Center, Eye Institute of Xiamen University, School of Medicine, Xiamen University, Xiamen 3611002, Fujian Province, China; Fujian Provincial Key Laboratory of Ophthalmology and Visual Science, Fujian Engineering and Research Center of Eye Regenerative Medicine, Xiamen 3611002, Fujian Province, ChinaDepartment of Traditional Chinese Medicine, School of Medicine, Xiamen University, Xiamen 361100, Fujian Province, ChinaDepartment of Ophthalmology, the First Affiliated Hospital of University of South China, Hengyang 421001, Hunan Province, ChinaDepartment of Traditional Chinese Medicine, School of Medicine, Xiamen University, Xiamen 361100, Fujian Province, ChinaXiamen University Affiliated Xiamen Eye Center, Eye Institute of Xiamen University, School of Medicine, Xiamen University, Xiamen 3611002, Fujian Province, China; Fujian Provincial Key Laboratory of Ophthalmology and Visual Science, Fujian Engineering and Research Center of Eye Regenerative Medicine, Xiamen 3611002, Fujian Province, ChinaXiamen University Affiliated Xiamen Eye Center, Eye Institute of Xiamen University, School of Medicine, Xiamen University, Xiamen 3611002, Fujian Province, China; Fujian Provincial Key Laboratory of Ophthalmology and Visual Science, Fujian Engineering and Research Center of Eye Regenerative Medicine, Xiamen 3611002, Fujian Province, ChinaXiamen University Affiliated Xiamen Eye Center, Eye Institute of Xiamen University, School of Medicine, Xiamen University, Xiamen 3611002, Fujian Province, China; Fujian Provincial Key Laboratory of Ophthalmology and Visual Science, Fujian Engineering and Research Center of Eye Regenerative Medicine, Xiamen 3611002, Fujian Province, ChinaXiamen University Affiliated Xiamen Eye Center, Eye Institute of Xiamen University, School of Medicine, Xiamen University, Xiamen 3611002, Fujian Province, China; The Second Affiliated Hospital, Hengyang Medical School, University of South China, Hengyang 421000, Hunan Province, China; Fujian Provincial Key Laboratory of Ophthalmology and Visual Science, Fujian Engineering and Research Center of Eye Regenerative Medicine, Xiamen 3611002, Fujian Province, China; Department of Traditional Chinese Medicine, School of Medicine, Xiamen University, Xiamen 361100, Fujian Province, ChinaAIM: To establish a stable, short-time, low-cost and reliable murine model of meibomian gland dysfunction (MGD). METHODS: A filter paper sheet soaked in 1.0 mol/L sodium hydroxide (NaOH) solution was used to touch the eyelid margin of C57BL/6J mice for 10s to establish the model. The other eye was left untreated as a control group. Eyelid margin morphological changes and the meibomian glands (MGs) were observed by slit lamp microscopy on days 5 and 10 post-burn. Hematoxylin-eosin (HE) staining and Oil red O staining were adopted in detecting the changes in MGs morphology and lipid deposition. Real-time polymerase chain reaction, Western blot, immunofluorescence staining and immunohistochemical staining were used to detect interleukin (IL)-6, IL-1β, IL-18, tumor necroses factor (TNF)-α, interferon (IFN)-γ, nicotinamide adenine dinucleotide phosphate (NADPH) oxidase 4 (NOX4), 3-nitroturosine (3-NT), 4-hydroxynonenal (4-HNE) and cytokeratin 10 (K10) expression changes in MGs. RESULTS: MGs showed plugging of orifice, glandular deficiency, abnormal acinar morphology, ductal dilatation, and lipid deposition after alkali burn. The expressions of IL-6, IL-18, IL-1β, IFN-γ, and TNF-α indicators of inflammation and oxidative stress in MGs tissues were significantly increased. Abnormal keratinization increased in the MG duct. CONCLUSION: A murine model of MGD is established by alkali burn of the eyelid margin that matches the clinical presentation of MGD providing a stable, short-time, low-cost, and reliable MGD model. The new method suggests efficient avenues for future research.http://ies.ijo.cn/en_publish/2024/12/20241202.pdfalkali burnmeibomian gland dysfunctionanimal modeloxidative stress
spellingShingle Yong Li
Ya-Qiong Yang
Yong Lin
Ke Yan
Yu-Fei Lyu
Zhao-Qiang Zhang
Cai-Hong Huang
Jiao-Yue Hu
Zu-Guo Liu
Alkali burn injury model of meibomian gland dysfunction in mice
International Journal of Ophthalmology
alkali burn
meibomian gland dysfunction
animal model
oxidative stress
title Alkali burn injury model of meibomian gland dysfunction in mice
title_full Alkali burn injury model of meibomian gland dysfunction in mice
title_fullStr Alkali burn injury model of meibomian gland dysfunction in mice
title_full_unstemmed Alkali burn injury model of meibomian gland dysfunction in mice
title_short Alkali burn injury model of meibomian gland dysfunction in mice
title_sort alkali burn injury model of meibomian gland dysfunction in mice
topic alkali burn
meibomian gland dysfunction
animal model
oxidative stress
url http://ies.ijo.cn/en_publish/2024/12/20241202.pdf
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